Showing results (1-10 of 12) with videos related to

Sort By:
Pageof 2
Experimental Cell Research|June 15, 2004
Adenylate kinase I does not affect cellular growth characteristics under normal and metabolic stress conditionsWieke de Bruin, Frank Oerlemans, Bé Wieringa
Plos Biology|March 14, 2008
Creatine kinase-mediated ATP supply fuels actin-based events in phagocytosisJan W P Kuiper, Helma Pluk, Frank Oerlemans, et al.
Behavioural Brain Research|January 11, 2005
Structural and behavioural consequences of double deficiency for creatine kinases BCK and UbCKmitFemke Streijger, Frank Oerlemans, Bart A Ellenbroek, et al.
Magnetic Resonance in Medicine|October 31, 2003
MR spectroscopy of muscle and brain in guanidinoacetate methyltransferase (GAMT)-deficient mice: validation of an animal model to study creatine deficiencyW Klaas Jan Renema, Andreas Schmidt, Jack J A van Asten, et al.
Epilepsia|July 24, 2009
Complete brain-type creatine kinase deficiency in mice blocks seizure activity and affects intracellular calcium kineticsFemke Streijger, Wim J J M Scheenen, Gilles van Luijtelaar, et al.
Molecular and Cellular Biochemistry|February 24, 2004
Mice lacking the UbCKmit isoform of creatine kinase reveal slower spatial learning acquisition, diminished exploration and habituation, and reduced acoustic startle reflex responsesFemke Streijger, Carolina R Jost, Frank Oerlemans, et al.
Physiology & Behavior|May 8, 2009
Mice lacking brain-type creatine kinase activity show defective thermoregulationFemke Streijger, Helma Pluk, Frank Oerlemans, et al.
Physiological Genomics|July 1, 2004
Mild impairment of motor nerve repair in mice lacking PTP-BL tyrosine phosphatase activityDerick G Wansink, Wilma Peters, Iris Schaafsma, et al.
The European Journal of Neuroscience|June 13, 2002
Creatine kinase B-driven energy transfer in the brain is important for habituation and spatial learning behaviour, mossy fibre field size and determination of seizure susceptibilityCarolina R Jost, Catharina E E M Van Der Zee, Henricus J A In 't Zandt, et al.
Molecular Cancer|August 4, 2009
Increased OXPHOS activity precedes rise in glycolytic rate in H-RasV12/E1A transformed fibroblasts that develop a Warburg phenotypeAd J C de Groof, Mariska M te Lindert, Michiel M T van Dommelen, et al.
Pageof 2