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Frank U Müller

Showing results (11-20 of 45) with videos related to

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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 20, 2007
Cardiomyocyte-specific inactivation of transcription factor CREB in miceMarek Matus, Geertje Lewin, Frank Stümpel, et al.
Journal of Molecular and Cellular Cardiology Plus|January 13, 2025
Myocardial overexpression of protein phosphatase 2A-B56α improves resistance against ischemia-reperfusion injuryJulius R Herting, Anna M Berg, Katarina Hadova, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 3, 2007
On the role of junctin in cardiac Ca2+ handling, contractility, and heart failureUlrich Gergs, Tobias Berndt, Jan Buskase, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 20, 2012
Modulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytesDana Kučerová, Hideo A Baba, Peter Bokník, et al.
Frontiers in Pharmacology|September 7, 2018
Distinct Occurrence of Proarrhythmic Afterdepolarizations in Atrial Versus Ventricular Cardiomyocytes: Implications for Translational Research on Atrial ArrhythmiaNils Bögeholz, Paul Pauls, Dirk G Dechering, et al.
Frontiers in Cardiovascular Medicine|October 23, 2023
Hypercontractile cardiac phenotype in mice overexpressing the regulatory subunit PR72 of protein phosphatase 2AJulius R Herting, Jule H König, Katarina Hadova, et al.
The Journal of Biological Chemistry|October 17, 2014
Cardiac function is regulated by B56α-mediated targeting of protein phosphatase 2A (PP2A) to contractile relevant substratesUwe Kirchhefer, Christiane Brekle, John Eskandar, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 25, 2007
Triadin is a critical determinant of cellular Ca cycling and contractility in the heartUwe Kirchhefer, Jan Klimas, Hideo A Baba, et al.
Naunyn-Schmiedeberg'S Archives of Pharmacology|May 26, 2018
Successful overexpression of wild-type inhibitor-2 of PP1 in cardiovascular cellsThorsten Krause, Stefanie Grote-Wessels, Felix Balzer, et al.
International Journal of Cardiology. Heart & Vasculature|January 9, 2023
Increased <i>in vivo</i> perpetuation of whole-heart ventricular arrhythmia in heterozygous Na<sup>+</sup>/Ca<sup>2+</sup> exchanger knockout miceNils Bögeholz, Vincent Knappe, Paul Pauls, et al.
Pageof 5

Showing results (11-20 of 45) with videos related to

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Pageof 5
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 20, 2007
Cardiomyocyte-specific inactivation of transcription factor CREB in miceMarek Matus, Geertje Lewin, Frank Stümpel, et al.
Journal of Molecular and Cellular Cardiology Plus|January 13, 2025
Myocardial overexpression of protein phosphatase 2A-B56α improves resistance against ischemia-reperfusion injuryJulius R Herting, Anna M Berg, Katarina Hadova, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 3, 2007
On the role of junctin in cardiac Ca2+ handling, contractility, and heart failureUlrich Gergs, Tobias Berndt, Jan Buskase, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 20, 2012
Modulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytesDana Kučerová, Hideo A Baba, Peter Bokník, et al.
Frontiers in Pharmacology|September 7, 2018
Distinct Occurrence of Proarrhythmic Afterdepolarizations in Atrial Versus Ventricular Cardiomyocytes: Implications for Translational Research on Atrial ArrhythmiaNils Bögeholz, Paul Pauls, Dirk G Dechering, et al.
Frontiers in Cardiovascular Medicine|October 23, 2023
Hypercontractile cardiac phenotype in mice overexpressing the regulatory subunit PR72 of protein phosphatase 2AJulius R Herting, Jule H König, Katarina Hadova, et al.
The Journal of Biological Chemistry|October 17, 2014
Cardiac function is regulated by B56α-mediated targeting of protein phosphatase 2A (PP2A) to contractile relevant substratesUwe Kirchhefer, Christiane Brekle, John Eskandar, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 25, 2007
Triadin is a critical determinant of cellular Ca cycling and contractility in the heartUwe Kirchhefer, Jan Klimas, Hideo A Baba, et al.
Naunyn-Schmiedeberg'S Archives of Pharmacology|May 26, 2018
Successful overexpression of wild-type inhibitor-2 of PP1 in cardiovascular cellsThorsten Krause, Stefanie Grote-Wessels, Felix Balzer, et al.
International Journal of Cardiology. Heart & Vasculature|January 9, 2023
Increased <i>in vivo</i> perpetuation of whole-heart ventricular arrhythmia in heterozygous Na<sup>+</sup>/Ca<sup>2+</sup> exchanger knockout miceNils Bögeholz, Vincent Knappe, Paul Pauls, et al.
Pageof 5