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Fred K Friedman

Showing results (11-20 of 17) with videos related to

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Frontiers in Bioscience : a Journal and Virtual Library|June 16, 2007
The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 proteinYongxia Qu, Victor Adler, Lara Izotova, et al.
Cancer Chemotherapy and Pharmacology|June 5, 2003
Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growthMecheal Kanovsky, Josef Michl, Georgia Botzolaki, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|July 2, 2013
Identification of 2-piperidone as a biomarker of CYP2E1 activity through metabolomic phenotypingJie Cheng, Chi Chen, Krausz W Kristopher, et al.
International Journal of Cancer|May 12, 2006
PNC-28, a p53-derived peptide that is cytotoxic to cancer cells, blocks pancreatic cancer cell growth in vivoJosef Michl, Bruce Scharf, Anna Schmidt, et al.
Annals of Clinical and Laboratory Science|March 5, 2008
Site-specific phosphorylation of raf in cells containing oncogenic ras-p21 is likely mediated by jun-N-terminal kinaseVictor Adler, Wilbur Bowne, Josef Michl, et al.
Biochemistry|August 10, 2005
Functional interactions of Raf and MEK with Jun-N-terminal kinase (JNK) result in a positive feedback loop on the oncogenic Ras signaling pathwayVictor Adler, Yongxia Qu, Steven J Smith, et al.
Oncogene|March 12, 2003
Preferential induction of necrosis in human breast cancer cells by a p53 peptide derived from the MDM2 binding siteTamara N Do, Ramon V Rosal, Lisa Drew, et al.
Pageof 2

Showing results (11-20 of 17) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 17 results.
Frontiers in Bioscience : a Journal and Virtual Library|June 16, 2007
The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 proteinYongxia Qu, Victor Adler, Lara Izotova, et al.
Cancer Chemotherapy and Pharmacology|June 5, 2003
Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growthMecheal Kanovsky, Josef Michl, Georgia Botzolaki, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|July 2, 2013
Identification of 2-piperidone as a biomarker of CYP2E1 activity through metabolomic phenotypingJie Cheng, Chi Chen, Krausz W Kristopher, et al.
International Journal of Cancer|May 12, 2006
PNC-28, a p53-derived peptide that is cytotoxic to cancer cells, blocks pancreatic cancer cell growth in vivoJosef Michl, Bruce Scharf, Anna Schmidt, et al.
Annals of Clinical and Laboratory Science|March 5, 2008
Site-specific phosphorylation of raf in cells containing oncogenic ras-p21 is likely mediated by jun-N-terminal kinaseVictor Adler, Wilbur Bowne, Josef Michl, et al.
Biochemistry|August 10, 2005
Functional interactions of Raf and MEK with Jun-N-terminal kinase (JNK) result in a positive feedback loop on the oncogenic Ras signaling pathwayVictor Adler, Yongxia Qu, Steven J Smith, et al.
Oncogene|March 12, 2003
Preferential induction of necrosis in human breast cancer cells by a p53 peptide derived from the MDM2 binding siteTamara N Do, Ramon V Rosal, Lisa Drew, et al.
Pageof 2