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Nature Biotechnology|September 10, 2014
Scientific rigor and the art of motorcycle maintenanceMarcus Munafò, Simon Noble, William J Browne, et al.Journal of Huntington'S Disease|July 27, 2014
Cognitive deficits in transgenic and knock-in HTT mice parallel those in Huntington's diseaseAndrew M Farrar, Carol A Murphy, Neil E Paterson, et al.Plos Currents|September 18, 2013
Increased Body Weight of the BAC HD Transgenic Mouse Model of Huntington's Disease Accounts for Some but Not All of the Observed HD-like Motor DeficitsAndrea E Kudwa, Liliana B Menalled, Stephen Oakeshott, et al.Plos One|August 8, 2013
SAHA enhances synaptic function and plasticity in vitro but has limited brain availability in vivo and does not impact cognitionJesse E Hanson, Hank La, Emile Plise, et al.Molecular Neurobiology|April 30, 2026
Micro- and Nanoplastics in the Human Brain: Mechanistic Plausibility, Translational Challenges, and Links to Neurological Disease TrendsFarshid Ghiyamihoor, Azam Asemi Rad, Parya Hassanifar, et al.Journal of Medicinal Chemistry|September 13, 2011
Discovery of isoxazole analogues of sazetidine-A as selective α4β2-nicotinic acetylcholine receptor partial agonists for the treatment of depressionJianhua Liu, Li-Fang Yu, J Brek Eaton, et al.Pharmacology Research & Perspectives|December 16, 2014
The potent and selective α4β2*/α6*-nicotinic acetylcholine receptor partial agonist 2-[5-[5-((S)Azetidin-2-ylmethoxy)-3-pyridinyl]-3-isoxazolyl]ethanol demonstrates antidepressive-like behavior in animal models and a favorable ADME-tox profileLi-Fang Yu, J Brek Eaton, Han-Kun Zhang, et al.Plos One|January 4, 2013
Comprehensive behavioral and molecular characterization of a new knock-in mouse model of Huntington's disease: zQ175Liliana B Menalled, Andrea E Kudwa, Sam Miller, et al.Nature Biotechnology|July 5, 2016
Large-scale phenome analysis defines a behavioral signature for Huntington's disease genotype in miceVadim Alexandrov, Dani Brunner, Liliana B Menalled, et al.Plos One|June 24, 2014
Genetic deletion of transglutaminase 2 does not rescue the phenotypic deficits observed in R6/2 and zQ175 mouse models of Huntington's diseaseLiliana B Menalled, Andrea E Kudwa, Steve Oakeshott, et al.Pageof 12