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The Journal of Gene Medicine|November 16, 2004
PEGylated adenovirus vectors containing RGD peptides on the tip of PEG show high transduction efficiency and antibody evasion abilityYusuke Eto, Jian-Qing Gao, Fumiko Sekiguchi, et al.
Bioorganic & Medicinal Chemistry Letters|January 25, 2005
Design and synthesis of a peptide-PEG transporter tool for carrying adenovirus vector into cellsMitsuko Maeda, Shinya Kida, Keiko Hojo, et al.
Bioorganic & Medicinal Chemistry|July 23, 2018
Design and synthesis of novel anti-hyperalgesic agents based on 6-prenylnaringenin as the T-type calcium channel blockersHuy Du Nguyen, Takuya Okada, Shun Kitamura, et al.
Journal of Pharmacological Sciences|February 18, 2022
Development of hepatic impairment aggravates chemotherapy-induced peripheral neuropathy following oxaliplatin treatment: Evidence from clinical and preclinical studiesTomoyoshi Miyamoto, Risa Domoto, Fumiko Sekiguchi, et al.
Biochemical and Biophysical Research Communications|October 18, 2011
Involvement of ERK in NMDA receptor-independent cortical neurotoxicity of hydrogen sulfideYuko Kurokawa, Fumiko Sekiguchi, Satoko Kubo, et al.
European Journal of Pharmacology|September 19, 2020
Essential role of Cav3.2 T-type calcium channels in butyrate-induced colonic pain and nociceptor hypersensitivity in miceMaho Tsubota, Kazuki Matsui, Maki Nakano, et al.
The Journal of Pharmacology and Experimental Therapeutics|August 27, 2005
Signal transduction for proteinase-activated receptor-2-triggered prostaglandin E2 formation in human lung epithelial cellsNaoyuki Kawao, Mami Nagataki, Keita Nagasawa, et al.
European Journal of Pharmacology|April 24, 2025
HMGB1 derived from macrophages and enteric glial cells contributes to the butyrate-induced colonic hypersensitivity in miceMaho Tsubota, Kana Sasaki, Eunkyung Shin, et al.
Redox Biology|December 23, 2022
A hydrolysate of poly-trans-[(2-carboxyethyl)germasesquioxane] (Ge-132) suppresses Cav3.2-dependent pain by sequestering exogenous and endogenous sulfideFumiko Sekiguchi, Nene Koike, Yasuhiro Shimada, et al.
Journal of Pharmacological Sciences|September 8, 2019
Genetic deletion of Cav3.2 T-type calcium channels abolishes H2S-dependent somatic and visceral pain signaling in C57BL/6 miceKazuki Matsui, Maho Tsubota, Saaya Fukushi, et al.
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