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Cancer Research
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July 26, 2000
Exisulind induction of apoptosis involves guanosine 3',5'-cyclic monophosphate phosphodiesterase inhibition, protein kinase G activation, and attenuated beta-catenin
W J Thompson, G A Piazza, H Li, et al.
Cancer Research
|
January 15, 1997
Sulfone metabolite of sulindac inhibits mammary carcinogenesis
H J Thompson, C Jiang, J Lu, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research
|
October 29, 2000
Cyclic GMP mediates apoptosis induced by sulindac derivatives via activation of c-Jun NH2-terminal kinase 1
J W Soh, Y Mao, M G Kim, et al.
Cancer Research
|
June 15, 1997
Apoptosis primarily accounts for the growth-inhibitory properties of sulindac metabolites and involves a mechanism that is independent of cyclooxygenase inhibition, cell cycle arrest, and p53 induction
G A Piazza, A K Rahm, T S Finn, et al.
Cell Death and Differentiation
|
October 8, 2011
MiR-181 mediates cell differentiation by interrupting the Lin28 and let-7 feedback circuit
X Li, J Zhang, L Gao, et al.
Breast Cancer Research and Treatment
|
May 23, 1998
Effects of sulindac and its metabolites on growth and apoptosis in human mammary epithelial and breast carcinoma cell lines
E K Han, N Arber, H Yamamoto, et al.
Journal of the National Cancer Institute
|
August 16, 1995
Inhibition of mammary carcinogenesis in rats by sulfone metabolite of sulindac
H J Thompson, S Briggs, N S Paranka, et al.
Urology
|
February 5, 1999
Exisulind (sulindac sulfone) suppresses growth of human prostate cancer in a nude mouse xenograft model by increasing apoptosis
E T Goluboff, A Shabsigh, J A Saidi, et al.
Cancer Research
|
July 15, 1995
Antineoplastic drugs sulindac sulfide and sulfone inhibit cell growth by inducing apoptosis
G A Piazza, A L Rahm, M Krutzsch, et al.
International Journal of Tissue Reactions
|
January 23, 1999
Inhibition of angiogenesis by sulindac and its sulfone metabolite (FGN-1): a potential mechanism for their antineoplastic properties
E Skopińska-Rózewska, G A Piazza, E Sommer, et al.
Page
of 3
Search research articles
Search
Showing results (11-20 of 27) with videos related to
Sort By:
Page
of 3
Cancer Research
|
July 26, 2000
Exisulind induction of apoptosis involves guanosine 3',5'-cyclic monophosphate phosphodiesterase inhibition, protein kinase G activation, and attenuated beta-catenin
W J Thompson, G A Piazza, H Li, et al.
Cancer Research
|
January 15, 1997
Sulfone metabolite of sulindac inhibits mammary carcinogenesis
H J Thompson, C Jiang, J Lu, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research
|
October 29, 2000
Cyclic GMP mediates apoptosis induced by sulindac derivatives via activation of c-Jun NH2-terminal kinase 1
J W Soh, Y Mao, M G Kim, et al.
Cancer Research
|
June 15, 1997
Apoptosis primarily accounts for the growth-inhibitory properties of sulindac metabolites and involves a mechanism that is independent of cyclooxygenase inhibition, cell cycle arrest, and p53 induction
G A Piazza, A K Rahm, T S Finn, et al.
Cell Death and Differentiation
|
October 8, 2011
MiR-181 mediates cell differentiation by interrupting the Lin28 and let-7 feedback circuit
X Li, J Zhang, L Gao, et al.
Breast Cancer Research and Treatment
|
May 23, 1998
Effects of sulindac and its metabolites on growth and apoptosis in human mammary epithelial and breast carcinoma cell lines
E K Han, N Arber, H Yamamoto, et al.
Journal of the National Cancer Institute
|
August 16, 1995
Inhibition of mammary carcinogenesis in rats by sulfone metabolite of sulindac
H J Thompson, S Briggs, N S Paranka, et al.
Urology
|
February 5, 1999
Exisulind (sulindac sulfone) suppresses growth of human prostate cancer in a nude mouse xenograft model by increasing apoptosis
E T Goluboff, A Shabsigh, J A Saidi, et al.
Cancer Research
|
July 15, 1995
Antineoplastic drugs sulindac sulfide and sulfone inhibit cell growth by inducing apoptosis
G A Piazza, A L Rahm, M Krutzsch, et al.
International Journal of Tissue Reactions
|
January 23, 1999
Inhibition of angiogenesis by sulindac and its sulfone metabolite (FGN-1): a potential mechanism for their antineoplastic properties
E Skopińska-Rózewska, G A Piazza, E Sommer, et al.
Page
of 3