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Neurology|April 1, 1998
PCR-based strategy for the diagnosis of hereditary neuropathy with liability to pressure palsies and Charcot-Marie-Tooth disease type 1AP Young, F Stögbauer, H Wiebusch, et al.
International Journal of Cardiology|December 6, 1996
Interleukin-6 correlates with hemodynamic impairment during dobutamine administration in chronic heart failureM C Deng, M Erren, A Lütgen, et al.
Thrombosis and Haemostasis|May 9, 2001
High density lipoproteins induce cell cycle entry in vascular smooth muscle cells via mitogen activated protein kinase-dependent pathwayJ R Nofer, R Junker, E Pulawski, et al.
Clinical Chemistry|April 1, 1995
Smith-Lemli-Opitz syndrome diagnosed by using time-of-flight secondary-ion mass spectrometryU Seedorf, M Fobker, R Voss, et al.
Human Genetics|February 1, 1996
A missense mutation (Thr-6Pro) in the lysosomal acid lipase (LAL) gene is present with a high frequency in three different ethnic populations: impact on serum lipoprotein concentrationsS Muntoni, H Wiebusch, H Funke, et al.
The Journal of Biological Chemistry|February 5, 1993
Interaction of reconstituted high density lipoprotein discs containing human apolipoprotein A-I (ApoA-I) variants with murine adipocytes and macrophages. Evidence for reduced cholesterol efflux promotion by apoA-I(Pro165-->Arg)A von Eckardstein, G Castro, I Wybranska, et al.
European Heart Journal|January 29, 2002
Association of the GPIa C807T and GPIIIa PlA1/A2 polymorphisms with premature myocardial infarction in menG Benze, J Heinrich, H Schulte, et al.
Transplantation|November 27, 1995
The relation of interleukin-6, tumor necrosis factor-alpha, IL-2, and IL-2 receptor levels to cellular rejection, allograft dysfunction, and clinical events early after cardiac transplantationM C Deng, M Erren, L Kammerling, et al.
Acta Physiologica Scandinavica|October 2, 2002
Associations of HDL phospholipids and paraoxonase activity with coronary heart disease in postmenopausal womenM J Horter, S Sondermann, H Reinecke, et al.
Circulation|October 1, 1996
Compound heterozygosity for a structural apolipoprotein A-I variant, apo A-I(L141R)Pisa, and an apolipoprotein A-I null allele in patients with absence of HDL cholesterol, corneal opacifications, and coronary heart diseaseR Miccoli, A Bertolotto, R Navalesi, et al.
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