G Attardi|M Yoneda|A Chomyn - JoVE Visualize

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Biochimica Et Biophysica Acta|May 24, 1995

Complementation and segregation behavior of disease-causing mitochondrial DNA mutations in cellular model systemsG Attardi, M Yoneda, A Chomyn

Proceedings of the National Academy of Sciences of the United States of America|December 1, 1992

Marked replicative advantage of human mtDNA carrying a point mutation that causes the MELAS encephalomyopathyM Yoneda, A Chomyn, A Martinuzzi, et al.

Nucleic Acids Research|February 25, 1981

Alignment of the amino terminal amino acid sequence of human cytochrome c oxidase subunits I and II with the sequence of their putative mRNAsA Chomyn, M W Hunkapiller, G Attardi

Nature Genetics|May 1, 1995

MtDNA mutation in MERRF syndrome causes defective aminoacylation of tRNA(Lys) and premature translation terminationJ A Enriquez, A Chomyn, G Attardi

Nucleic Acids Research|January 16, 1999

Search for differences in post-transcriptional modification patterns of mitochondrial DNA-encoded wild-type and mutant human tRNALys and tRNALeu(UUR)M Helm, C Florentz, A Chomyn, et al.

The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 7, 1997

Myoclonic epilepsy and ragged red fibers (MERRF) syndrome: selective vulnerability of CNS neurons does not correlate with the level of mitochondrial tRNAlys mutation in individual neuronal isolatesL Zhou, A Chomyn, G Attardi, et al.

The Journal of Biological Chemistry|March 5, 1986

Antibodies against the COOH-terminal undecapeptide of subunit II, but not those against the NH2-terminal decapeptide, immunoprecipitate the whole human cytochrome c oxidase complexP Mariottini, A Chomyn, R F Doolittle, et al.

Muscle & Nerve. Supplement|January 1, 1995

Heteroplasmic mitochondrial tRNA(Lys) mutation and its complementation in MERRF patient-derived mitochondrial transformantsM Yoneda, T Miyatake, G Attardi

Molecular and Cellular Biology|April 1, 1994

Complementation of mutant and wild-type human mitochondrial DNAs coexisting since the mutation event and lack of complementation of DNAs introduced separately into a cell within distinct organellesM Yoneda, T Miyatake, G Attardi

Proceedings of the National Academy of Sciences of the United States of America|May 15, 1992

MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstream mature transcriptsA Chomyn, A Martinuzzi, M Yoneda, et al.

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Biochimica Et Biophysica Acta|May 24, 1995

Complementation and segregation behavior of disease-causing mitochondrial DNA mutations in cellular model systemsG Attardi, M Yoneda, A Chomyn

Proceedings of the National Academy of Sciences of the United States of America|December 1, 1992

Marked replicative advantage of human mtDNA carrying a point mutation that causes the MELAS encephalomyopathyM Yoneda, A Chomyn, A Martinuzzi, et al.

Nucleic Acids Research|February 25, 1981

Alignment of the amino terminal amino acid sequence of human cytochrome c oxidase subunits I and II with the sequence of their putative mRNAsA Chomyn, M W Hunkapiller, G Attardi

Nature Genetics|May 1, 1995

MtDNA mutation in MERRF syndrome causes defective aminoacylation of tRNA(Lys) and premature translation terminationJ A Enriquez, A Chomyn, G Attardi

Nucleic Acids Research|January 16, 1999

Search for differences in post-transcriptional modification patterns of mitochondrial DNA-encoded wild-type and mutant human tRNALys and tRNALeu(UUR)M Helm, C Florentz, A Chomyn, et al.

The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 7, 1997

Myoclonic epilepsy and ragged red fibers (MERRF) syndrome: selective vulnerability of CNS neurons does not correlate with the level of mitochondrial tRNAlys mutation in individual neuronal isolatesL Zhou, A Chomyn, G Attardi, et al.

The Journal of Biological Chemistry|March 5, 1986

Antibodies against the COOH-terminal undecapeptide of subunit II, but not those against the NH2-terminal decapeptide, immunoprecipitate the whole human cytochrome c oxidase complexP Mariottini, A Chomyn, R F Doolittle, et al.

Muscle & Nerve. Supplement|January 1, 1995

Heteroplasmic mitochondrial tRNA(Lys) mutation and its complementation in MERRF patient-derived mitochondrial transformantsM Yoneda, T Miyatake, G Attardi

Molecular and Cellular Biology|April 1, 1994

Complementation of mutant and wild-type human mitochondrial DNAs coexisting since the mutation event and lack of complementation of DNAs introduced separately into a cell within distinct organellesM Yoneda, T Miyatake, G Attardi

Proceedings of the National Academy of Sciences of the United States of America|May 15, 1992

MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstream mature transcriptsA Chomyn, A Martinuzzi, M Yoneda, et al.

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