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Biochemical Pharmacology|September 15, 1988
Alterations of surface morphology caused by the metabolism of menadione in mammalian cells are associated with the oxidation of critical sulfhydryl groups in cytoskeletal proteinsF Mirabelli, A Salis, M Perotti, et al.Toxicology|July 3, 1989
On the role of mitochondria in cell injury caused by vanadate-induced Ca2+ overloadP Richelmi, F Mirabelli, A Salis, et al.Archives of Biochemistry and Biophysics|May 1, 1989
Cytoskeletal alterations in human platelets exposed to oxidative stress are mediated by oxidative and Ca2+-dependent mechanismsF Mirabelli, A Salis, M Vairetti, et al.Free Radical Research Communications|January 1, 1990
The cytoskeleton as a target in quinone toxicityG Bellomo, F Mirabelli, P Richelmi, et al.Archives of Biochemistry and Biophysics|November 1, 1988
Alterations in hepatocyte cytoskeleton caused by redox cycling and alkylating quinonesH Thor, F Mirabelli, A Salis, et al.Archives of Biochemistry and Biophysics|July 1, 1988
Menadione-induced bleb formation in hepatocytes is associated with the oxidation of thiol groups in actinF Mirabelli, A Salis, V Marinoni, et al.Cancer Research|March 1, 1992
Tumor necrosis factor alpha induces apoptosis in mammary adenocarcinoma cells by an increase in intranuclear free Ca2+ concentration and DNA fragmentationG Bellomo, M Perotti, F Taddei, et al.Acta Diabetologica Latina|January 1, 1985
Insulin degradation in human erythrocyte: effects of cationsG Bellomo, P L Nicotera, F Travaglino, et al.Biochemical Pharmacology|April 15, 1987
Formation and reduction of glutathione-protein mixed disulfides during oxidative stress. A study with isolated hepatocytes and menadione (2-methyl-1,4-naphthoquinone)G Bellomo, F Mirabelli, D DiMonte, et al.Biochemical Pharmacology|October 15, 1985
On the role of thiol groups in the inhibition of liver microsomal Ca2+ sequestration by toxic agentsH Thor, P Hartzell, S A Svensson, et al.Pageof 26