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European Journal of Cancer (Oxford, England : 1990)|March 23, 2021
Lost in application: Measuring hypoxia for radiotherapy optimisationNiluja Thiruthaneeswaran, Becky A S Bibby, Lingjang Yang, et al.
Cancer Chemotherapy and Pharmacology|April 6, 2017
Up-regulation of autophagy is a mechanism of resistance to chemotherapy and can be inhibited by pantoprazole to increase drug sensitivityQian Tan, A M Joshua, M Wang, et al.
Molecular Cancer Therapeutics|January 14, 2009
Targeting homologous recombination using imatinib results in enhanced tumor cell chemosensitivity and radiosensitivityAnanya Choudhury, Helen Zhao, Farid Jalali, et al.
Radiation Research|November 1, 2014
Hypoxia and cellular localization influence the radiosensitizing effect of gold nanoparticles (AuNPs) in breast cancer cellsLei Cui, Kenneth Tse, Payam Zahedi, et al.
International Journal of Radiation Oncology, Biology, Physics|July 10, 2010
Ionizing radiation activates AMP-activated kinase (AMPK): a target for radiosensitization of human cancer cellsToran Sanli, Ayesha Rashid, Caiqiong Liu, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|August 27, 2009
Longitudinal cytokine expression during IMRT for prostate cancer and acute treatment toxicityEva Christensen, Melania Pintilie, Kenneth R Evans, et al.
Cancer Research|October 14, 2010
Tumor cell kill by c-MYC depletion: role of MYC-regulated genes that control DNA double-strand break repairKaisa R Luoto, Alice X Meng, Amanda R Wasylishen, et al.
Radiation Research|January 14, 2017
Significant Radiation Enhancement Effects by Gold Nanoparticles in Combination with Cisplatin in Triple Negative Breast Cancer Cells and Tumor XenograftsLei Cui, Sohyoung Her, Michael Dunne, et al.
ACS Nano|April 3, 2013
Inherently multimodal nanoparticle-driven tracking and real-time delineation of orthotopic prostate tumors and micrometastasesTracy W Liu, Thomas D Macdonald, Cheng S Jin, et al.
The Journal of Cell Biology|October 13, 2006
Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATRGraham Dellaire, Reagan W Ching, Kashif Ahmed, et al.
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