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G Butler

Showing results (901-910 of 1,112) with videos related to

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Annals of the New York Academy of Sciences|July 23, 1999
Evaluation of some newer matrix metalloproteinasesG Murphy, V Knäuper, S Cowell, et al.
Clinical Dysmorphology|April 20, 2001
Total anomalous pulmonary venous connection and a constellation of craniofacial, skeletal, and urogenital anomalies in a newborn and similar features in his 36-year-old fatherD M Pierson, E M Taboada, G K Lofland, et al.
American Journal of Medical Genetics|July 11, 1997
Hypopigmentation in the Prader-Willi syndrome correlates with P gene deletion but not with haplotype of the hemizygous P alleleR A Spritz, T Bailin, R D Nicholls, et al.
Pediatrics|February 1, 1993
Prader-Willi syndrome: consensus diagnostic criteriaV A Holm, S B Cassidy, M G Butler, et al.
Nature Communications|August 25, 2022
Destabilisation of the Subpolar North Atlantic prior to the Little Ice AgeBeatriz Arellano-Nava, Paul R Halloran, Chris A Boulton, et al.
Endocrinology|April 24, 2001
Responses of the fetal pituitary-adrenal axis to acute and chronic hypoglycemia during late gestation in the sheepL J Edwards, M E Symonds, K E Warnes, et al.
Health Technology Assessment (Winchester, England)|June 21, 2007
A systematic review of the routine monitoring of growth in children of primary school age to identify growth-related conditionsD Fayter, J Nixon, S Hartley, et al.
American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|January 14, 2012
The neuroanatomy of genetic subtype differences in Prader-Willi syndromeRobyn A Honea, Laura M Holsen, Rebecca J Lepping, et al.
American Journal of Medical Genetics. Part A|January 19, 2008
Expanding the phenotype of SPONASTRIME dysplasia to include short dental roots, hypogammaglobulinemia, and cataractsKaren W Gripp, Caitlyn Johnson, Charles I Scott, et al.
Chest|June 1, 1997
The presence of genetic anticipation suggests that the molecular basis of familial primary pulmonary hypertension may be trinucleotide repeat expansionJ E Loyd, B Slovis, J A Phillips, et al.
Pageof 112

Showing results (901-910 of 1,112) with videos related to

Sort By:
Pageof 112
Annals of the New York Academy of Sciences|July 23, 1999
Evaluation of some newer matrix metalloproteinasesG Murphy, V Knäuper, S Cowell, et al.
Clinical Dysmorphology|April 20, 2001
Total anomalous pulmonary venous connection and a constellation of craniofacial, skeletal, and urogenital anomalies in a newborn and similar features in his 36-year-old fatherD M Pierson, E M Taboada, G K Lofland, et al.
American Journal of Medical Genetics|July 11, 1997
Hypopigmentation in the Prader-Willi syndrome correlates with P gene deletion but not with haplotype of the hemizygous P alleleR A Spritz, T Bailin, R D Nicholls, et al.
Pediatrics|February 1, 1993
Prader-Willi syndrome: consensus diagnostic criteriaV A Holm, S B Cassidy, M G Butler, et al.
Nature Communications|August 25, 2022
Destabilisation of the Subpolar North Atlantic prior to the Little Ice AgeBeatriz Arellano-Nava, Paul R Halloran, Chris A Boulton, et al.
Endocrinology|April 24, 2001
Responses of the fetal pituitary-adrenal axis to acute and chronic hypoglycemia during late gestation in the sheepL J Edwards, M E Symonds, K E Warnes, et al.
Health Technology Assessment (Winchester, England)|June 21, 2007
A systematic review of the routine monitoring of growth in children of primary school age to identify growth-related conditionsD Fayter, J Nixon, S Hartley, et al.
American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|January 14, 2012
The neuroanatomy of genetic subtype differences in Prader-Willi syndromeRobyn A Honea, Laura M Holsen, Rebecca J Lepping, et al.
American Journal of Medical Genetics. Part A|January 19, 2008
Expanding the phenotype of SPONASTRIME dysplasia to include short dental roots, hypogammaglobulinemia, and cataractsKaren W Gripp, Caitlyn Johnson, Charles I Scott, et al.
Chest|June 1, 1997
The presence of genetic anticipation suggests that the molecular basis of familial primary pulmonary hypertension may be trinucleotide repeat expansionJ E Loyd, B Slovis, J A Phillips, et al.
Pageof 112