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Oncogene|December 24, 1997
Upregulation of the angiogenic factors PlGF, VEGF and their receptors (Flt-1, Flk-1/KDR) by TSH in cultured thyrocytes and in the thyroid gland of thiouracil-fed rats suggest a TSH-dependent paracrine mechanism for goiter hypervascularizationG Viglietto, A Romano, G Manzo, et al.
Endocrine-Related Cancer|May 27, 2006
MicroRNA deregulation in human thyroid papillary carcinomasP Pallante, R Visone, M Ferracin, et al.
International Journal of Cancer|January 9, 2001
High mobility group HMGI(Y) protein expression in human colorectal hyperplastic and neoplastic diseasesG Chiappetta, G Manfioletti, F Pentimalli, et al.
Oncogene|August 5, 1998
Increase in AP-1 activity is a general event in thyroid cell transformation in vitro and in vivoS Battista, F de Nigris, M Fedele, et al.
Oncogene|August 1, 1996
Neovascularization in human germ cell tumors correlates with a marked increase in the expression of the vascular endothelial growth factor but not the placenta-derived growth factorG Viglietto, A Romano, D Maglione, et al.
Molecular and Cellular Biology|March 22, 2001
Critical role of the HMGI(Y) proteins in adipocytic cell growth and differentiationR M Melillo, G M Pierantoni, S Scala, et al.
Cancer Research|September 29, 1998
Detection of high mobility group I HMGI(Y) protein in the diagnosis of thyroid tumors: HMGI(Y) expression represents a potential diagnostic indicator of carcinomaG Chiappetta, G Tallini, M C De Biasio, et al.
Blood|April 15, 1997
Expression of the RET receptor tyrosine kinase and GDNFR-alpha in normal and leukemic human hematopoietic cells and stromal cells of the bone marrow microenvironmentV Gattei, A Celetti, A Cerrato, et al.
The Journal of Clinical Endocrinology and Metabolism|November 24, 1999
High prevalence of RET/PTC rearrangements in Ukrainian and Belarussian post-Chernobyl thyroid papillary carcinomas: a strong correlation between RET/PTC3 and the solid-follicular variantG A Thomas, H Bunnell, H A Cook, et al.
Oncogene|June 21, 2006
Thyroid targeting of the N-ras(Gln61Lys) oncogene in transgenic mice results in follicular tumors that progress to poorly differentiated carcinomasD Vitagliano, G Portella, G Troncone, et al.
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