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Cardiovascular Research
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March 1, 1991
Frequency dependent effects of tocainide, quinidine, and flecainide on conduction as reflected in the rise time of the monophasic action potential in the isolated guinea pig heart
B G Duker
Journal of Pharmacological Methods
|
November 1, 1987
Recording of monophasic action potentials in the horizontally perfused guinea pig heart. An in vitro method to study drug-induced changes in cardiac electrophysiology
J J Reid, G Duker
Journal of Cardiovascular Electrophysiology
|
November 1, 1996
Lidocaine and nisoldipine attenuate almokalant-induced dispersion of repolarization and early afterdepolarizations in vitro
C Abrahamsson, L Carlsson, G Duker
The Journal of Pharmacology and Experimental Therapeutics
|
October 1, 1996
Rhythm anomalies related to delayed repolarization in vivo: influence of sarcolemmal Ca++ entry and intracellular Ca++ overload
L Carlsson, L Drews, G Duker
Pharmacology & Toxicology
|
August 1, 1988
Computerized evaluation of drug-induced changes in guinea-pig epicardial monophasic action potentials
G Duker, O Almgren, J Axenborg
Journal of Cardiovascular Pharmacology
|
September 1, 1992
Electrophysiologic and hemodynamic effects of H 234/09 (almokalant), quinidine, and (+)-sotalol in the anesthetized dog
G Duker, O Almgren, L Carlsson
Journal of Cardiovascular Pharmacology
|
August 1, 1990
QTU-prolongation and torsades de pointes induced by putative class III antiarrhythmic agents in the rabbit: etiology and interventions
L Carlsson, O Almgren, G Duker
Basic Research in Cardiology
|
January 1, 1987
Effects of nifedipine on collateral blood flow at the lateral border of the acutely ischemic myocardium
P O Sjöquist, G Duker, I Hirsch
Journal of Cardiovascular Pharmacology
|
July 1, 1985
Coronary collateral blood flow in acute myocardial ischemia is not increased by dihydropyridine-induced coronary vasodilatation
P O Sjöquist, G Duker, O Almgren
Circulation
|
April 18, 2001
Instability and triangulation of the action potential predict serious proarrhythmia, but action potential duration prolongation is antiarrhythmic
L M Hondeghem, L Carlsson, G Duker
Page
of 3
Search research articles
Search
Showing results (1-10 of 27) with videos related to
Sort By:
Page
of 3
Cardiovascular Research
|
March 1, 1991
Frequency dependent effects of tocainide, quinidine, and flecainide on conduction as reflected in the rise time of the monophasic action potential in the isolated guinea pig heart
B G Duker
Journal of Pharmacological Methods
|
November 1, 1987
Recording of monophasic action potentials in the horizontally perfused guinea pig heart. An in vitro method to study drug-induced changes in cardiac electrophysiology
J J Reid, G Duker
Journal of Cardiovascular Electrophysiology
|
November 1, 1996
Lidocaine and nisoldipine attenuate almokalant-induced dispersion of repolarization and early afterdepolarizations in vitro
C Abrahamsson, L Carlsson, G Duker
The Journal of Pharmacology and Experimental Therapeutics
|
October 1, 1996
Rhythm anomalies related to delayed repolarization in vivo: influence of sarcolemmal Ca++ entry and intracellular Ca++ overload
L Carlsson, L Drews, G Duker
Pharmacology & Toxicology
|
August 1, 1988
Computerized evaluation of drug-induced changes in guinea-pig epicardial monophasic action potentials
G Duker, O Almgren, J Axenborg
Journal of Cardiovascular Pharmacology
|
September 1, 1992
Electrophysiologic and hemodynamic effects of H 234/09 (almokalant), quinidine, and (+)-sotalol in the anesthetized dog
G Duker, O Almgren, L Carlsson
Journal of Cardiovascular Pharmacology
|
August 1, 1990
QTU-prolongation and torsades de pointes induced by putative class III antiarrhythmic agents in the rabbit: etiology and interventions
L Carlsson, O Almgren, G Duker
Basic Research in Cardiology
|
January 1, 1987
Effects of nifedipine on collateral blood flow at the lateral border of the acutely ischemic myocardium
P O Sjöquist, G Duker, I Hirsch
Journal of Cardiovascular Pharmacology
|
July 1, 1985
Coronary collateral blood flow in acute myocardial ischemia is not increased by dihydropyridine-induced coronary vasodilatation
P O Sjöquist, G Duker, O Almgren
Circulation
|
April 18, 2001
Instability and triangulation of the action potential predict serious proarrhythmia, but action potential duration prolongation is antiarrhythmic
L M Hondeghem, L Carlsson, G Duker
Page
of 3