Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

G F Tomaselli

Showing results (51-60 of 68) with videos related to

Pageof 7
Sort By:
Circulation|March 12, 1998
Phenotypic characterization of a novel long-QT syndrome mutation (R1623Q) in the cardiac sodium channelN G Kambouris, H B Nuss, D C Johns, et al.
The Journal of Physiology|July 15, 1996
Coupling between fast and slow inactivation revealed by analysis of a point mutation (F1304Q) in mu 1 rat skeletal muscle sodium channelsH B Nuss, J R Balser, D W Orias, et al.
The Journal of Clinical Investigation|April 20, 2000
A revised view of cardiac sodium channel "blockade" in the long-QT syndromeN G Kambouris, H B Nuss, D C Johns, et al.
Journal of Cardiovascular Electrophysiology|October 24, 1998
Beat-to-beat repolarization lability identifies patients at risk for sudden cardiac deathW L Atiga, H Calkins, J H Lawrence, et al.
Gene Therapy|October 1, 1996
Reversal of potassium channel deficiency in cells from failing hearts by adenoviral gene transfer: a prototype for gene therapy for disorders of cardiac excitability and contractilityH B Nuss, D C Johns, S Kääb, et al.
Journal of the American College of Cardiology|August 1, 1997
Repolarization abnormalities, arrhythmia and sudden death in canine tachycardia-induced cardiomyopathyP H Pak, H B Nuss, R S Tunin, et al.
Biophysical Journal|September 1, 1996
Single-channel analysis of inactivation-defective rat skeletal muscle sodium channels containing the F1304Q mutationJ H Lawrence, D W Orias, J R Balser, et al.
Circulation Research|February 1, 1996
Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failureS Kääb, H B Nuss, N Chiamvimonvat, et al.
Circulation|October 7, 1998
Molecular basis of transient outward potassium current downregulation in human heart failure: a decrease in Kv4.3 mRNA correlates with a reduction in current densityS Kääb, J Dixon, J Duc, et al.
The Journal of Clinical Investigation|December 15, 1996
Local anesthetics as effectors of allosteric gating. Lidocaine effects on inactivation-deficient rat skeletal muscle Na channelsJ R Balser, H B Nuss, D W Orias, et al.
Pageof 7

Showing results (51-60 of 68) with videos related to

Sort By:
Pageof 7
Circulation|March 12, 1998
Phenotypic characterization of a novel long-QT syndrome mutation (R1623Q) in the cardiac sodium channelN G Kambouris, H B Nuss, D C Johns, et al.
The Journal of Physiology|July 15, 1996
Coupling between fast and slow inactivation revealed by analysis of a point mutation (F1304Q) in mu 1 rat skeletal muscle sodium channelsH B Nuss, J R Balser, D W Orias, et al.
The Journal of Clinical Investigation|April 20, 2000
A revised view of cardiac sodium channel "blockade" in the long-QT syndromeN G Kambouris, H B Nuss, D C Johns, et al.
Journal of Cardiovascular Electrophysiology|October 24, 1998
Beat-to-beat repolarization lability identifies patients at risk for sudden cardiac deathW L Atiga, H Calkins, J H Lawrence, et al.
Gene Therapy|October 1, 1996
Reversal of potassium channel deficiency in cells from failing hearts by adenoviral gene transfer: a prototype for gene therapy for disorders of cardiac excitability and contractilityH B Nuss, D C Johns, S Kääb, et al.
Journal of the American College of Cardiology|August 1, 1997
Repolarization abnormalities, arrhythmia and sudden death in canine tachycardia-induced cardiomyopathyP H Pak, H B Nuss, R S Tunin, et al.
Biophysical Journal|September 1, 1996
Single-channel analysis of inactivation-defective rat skeletal muscle sodium channels containing the F1304Q mutationJ H Lawrence, D W Orias, J R Balser, et al.
Circulation Research|February 1, 1996
Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failureS Kääb, H B Nuss, N Chiamvimonvat, et al.
Circulation|October 7, 1998
Molecular basis of transient outward potassium current downregulation in human heart failure: a decrease in Kv4.3 mRNA correlates with a reduction in current densityS Kääb, J Dixon, J Duc, et al.
The Journal of Clinical Investigation|December 15, 1996
Local anesthetics as effectors of allosteric gating. Lidocaine effects on inactivation-deficient rat skeletal muscle Na channelsJ R Balser, H B Nuss, D W Orias, et al.
Pageof 7