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Cardiovascular Research|December 1, 1996
Low-dose inotropic stimulation during left ventricular ischaemia does not worsen post-ischaemic dysfunctionG G Schwartz, Y Xu, C Greyson, et al.Cardiovascular Research|May 1, 1997
Right ventricular dysfunction persists following brief right ventricular pressure overloadC Greyson, Y Xu, J Cohen, et al.American Journal of Physiology. Heart and Circulatory Physiology|April 25, 2000
Right ventricular pressure and dilation during pressure overload determine dysfunction after pressure overloadC Greyson, Y Xu, L Lu, et al.American Journal of Physiology. Heart and Circulatory Physiology|March 15, 2001
A common mechanism for concurrent changes of diastolic muscle length and systolic function in intact heartsL Lu, Y Xu, P Zhu, et al.American Journal of Physiology. Heart and Circulatory Physiology|February 9, 2000
Glucose-insulin-potassium preserves systolic and diastolic function in ischemia and reperfusion in pigsP Zhu, L Lu, Y Xu, et al.The American Journal of Physiology|December 9, 1998
beta-adrenergic stimulation induces transient imbalance between myocardial substrate uptake and metabolism in vivoY Xu, L Lu, P Zhu, et al.Circulation|March 15, 2000
Troglitazone improves recovery of left ventricular function after regional ischemia in pigsP Zhu, L Lu, Y Xu, et al.The American Journal of Physiology|May 1, 1995
Effects of inotropic stimulation on energy metabolism and systolic function of ischemic right ventricleC Greyson, J Garcia, M Mayr, et al.The American Journal of Physiology|July 1, 1994
Inhibition of fatty acid metabolism alters myocardial high-energy phosphates in vivoG G Schwartz, C Greyson, J A Wisneski, et al.Journal of Molecular and Cellular Cardiology|May 25, 1999
Non-elastic deformation of myocardium in low-flow ischemia and reperfusion: ultrastructure-function relationsL Lu, Y Xu, C R Greyson, et al.Pageof 1,308