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Molecular Systems Biology|June 3, 2018
Forces driving the three-dimensional folding of eukaryotic genomesAlvaro Rada-Iglesias, Frank G Grosveld, Argyris PapantonisMethods (San Diego, Calif.)|September 18, 2001
Fluorescence in situ hybridization analysis of transcript dynamics in cellsM P van de Corput, F G GrosveldProceedings of the National Academy of Sciences of the United States of America|March 4, 1997
DEK, an autoantigen involved in a chromosomal translocation in acute myelogenous leukemia, binds to the HIV-2 enhancerG K Fu, G Grosveld, D M MarkovitzProceedings of the National Academy of Sciences of the United States of America|January 1, 1982
Isolation of transforming DNA by cosmid rescueT Lund, F G Grosveld, R A FlavellNeoplasia (New York, N.Y.)|August 10, 2000
Tel, a frequent target of leukemic translocations, induces cellular aggregation and influences expression of extracellular matrix componentsL Van Rompaey, W Dou, A Buijs, et al.The EMBO Journal|October 15, 1996
G2 arrest and impaired nucleocytoplasmic transport in mouse embryos lacking the proto-oncogene CAN/Nup214J van Deursen, J Boer, L Kasper, et al.Cancer Genetics and Cytogenetics|April 1, 1991
Review of clinical, cytogenetic, and molecular aspects of Ph-negative CMLD C van der Plas, G Grosveld, A HagemeijerJournal of Immunogenetics|February 1, 1990
Inactivation of the H-2Klk gene could involve the substitutions of methylated CpGsB C Sim, F G Grosveld, K M HuiOncogene|November 15, 2000
Tel induces a G1 arrest and suppresses Ras-induced transformationL V Rompaey, M Potter, C Adams, et al.Molecular and Cellular Biology|August 1, 1990
The (6;9) chromosome translocation, associated with a specific subtype of acute nonlymphocytic leukemia, leads to aberrant transcription of a target gene on 9q34M von Lindern, A Poustka, H Lerach, et al.Pageof 16