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Neuroscience Letters|January 9, 2013
Genetic analysis of the ATG7 gene promoter in sporadic Parkinson's diseaseDongfeng Chen, Shuchao Pang, Xungang Feng, et al.
Blood|July 29, 1998
Overexpression of HOX11 leads to the immortalization of embryonic precursors with both primitive and definitive hematopoietic potentialG Keller, C Wall, A Z Fong, et al.
Molecular and Cellular Biology|July 30, 2004
Hematopoietic cell fate and the initiation of leukemic properties in primitive primary human cells are influenced by Ras activity and farnesyltransferase inhibitionCraig Dorrell, Katsuto Takenaka, Mark D Minden, et al.
Journal of Immunological Methods|April 8, 1992
Increased frequency of both total and specific monoclonal antibody producing hybridomas using a fusion partner that constitutively expresses recombinant IL-6J F Harris, R G Hawley, T S Hawley, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|October 29, 2004
Sustained phenotypic correction of hemophilia a mice following oncoretroviral-mediated expression of a bioengineered human factor VIII gene in long-term hematopoietic repopulating cellsMorvarid Moayeri, Ali Ramezani, Richard A Morgan, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 1, 1982
Mutant immunoglobulin genes have repetitive DNA elements inserted into their intervening sequencesR G Hawley, M J Shulman, H Murialdo, et al.
Molecular Medicine Reports|February 6, 2019
Functional genetic variants of the GATA4 gene promoter in acute myocardial infarctionJing Chen, Shuai Wang, Shuchao Pang, et al.
Current Gene Therapy|January 30, 2003
Development of improved factor VIII molecules and new gene transfer approaches for hemophilia AEvgueni L Saenko, Natalya M Ananyeva, Morvarid Moayeri, et al.
The Journal of Biological Chemistry|May 2, 2003
BCL-2 and BCL-XL restrict lineage choice during hematopoietic differentiationLoralee Haughn, Robert G Hawley, Deborah K Morrison, et al.
Oncogene|April 4, 1996
Activation of the erythropoietin gene in the majority of F-MuLV-induced erythroleukemias results in growth factor independence and enhanced tumorigenicityJ C Howard, L Berger, M R Bani, et al.
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