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The EMBO Journal|April 15, 1996
The p38/RK mitogen-activated protein kinase pathway regulates interleukin-6 synthesis response to tumor necrosis factorR Beyaert, A Cuenda, W Vanden Berghe, et al.
The Journal of Biological Chemistry|May 23, 1998
The 55-kDa tumor necrosis factor receptor induces clustering of mitochondria through its membrane-proximal regionK De Vos, V Goossens, E Boone, et al.
The Journal of Cell Biology|June 29, 1999
The zinc finger protein A20 inhibits TNF-induced NF-kappaB-dependent gene expression by interfering with an RIP- or TRAF2-mediated transactivation signal and directly binds to a novel NF-kappaB-inhibiting protein ABINK Heyninck, D De Valck, W Vanden Berghe, et al.
British Journal of Pharmacology|May 30, 1998
Agonistic properties of alniditan, sumatriptan and dihydroergotamine on human 5-HT1B and 5-HT1D receptors expressed in various mammalian cell linesA S Lesage, R Wouters, P Van Gompel, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 23, 1999
Multiple signal transduction pathways regulate TNF-induced actin reorganization in macrophages: inhibition of Cdc42-mediated filopodium formation by TNFM Peppelenbosch, E Boone, G E Jones, et al.
Immunology Letters|December 1, 1987
Gene cloning and structure--function relationship of cytokines such as TNF and interleukinsW Fiers, R Beyaert, P Brouckaert, et al.
European Journal of Biochemistry|March 7, 2001
Structure-activity relationship of the p55 TNF receptor death domain and its lymphoproliferation mutantsG De Wilde, J Murray-Rust, E Boone, et al.
Cancer Research|June 1, 1993
Sensitization of tumor cells to tumor necrosis factor action by the protein kinase inhibitor staurosporineR Beyaert, B Vanhaesebroeck, K Heyninck, et al.
The Journal of Biological Chemistry|September 16, 2000
Structure/Function analysis of p55 tumor necrosis factor receptor and fas-associated death domain. Effect on necrosis in L929sA cellsE Boone, T Vanden Berghe, G Van Loo, et al.
The Journal of Biological Chemistry|November 5, 1999
Peroxisome proliferator-activated receptor alpha negatively regulates the vascular inflammatory gene response by negative cross-talk with transcription factors NF-kappaB and AP-1P Delerive, K De Bosscher, S Besnard, et al.
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