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G I Bell

Showing results (241-250 of 317) with videos related to

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Diabetes|February 1, 1996
Identification of microsatellite markers near the human genes encoding the beta-cell ATP-sensitive K+ channel and linkage studies with NIDDM in JapaneseN Iwasaki, M Kawamura, K Yamagata, et al.
The Journal of Clinical Investigation|April 1, 1989
Sequence of the variant thyroxine-binding globulin of Australian aborigines. Only one of two amino acid replacements is responsible for its altered propertiesK Takeda, Y Mori, S Sobieszczyk, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 1, 1988
Sequence, tissue distribution, and chromosomal localization of mRNA encoding a human glucose transporter-like proteinH Fukumoto, S Seino, H Imura, et al.
Molecular Endocrinology (Baltimore, Md.)|December 1, 1992
Somatostatin receptors, an expanding gene family: cloning and functional characterization of human SSTR3, a protein coupled to adenylyl cyclaseY Yamada, T Reisine, S F Law, et al.
The Journal of Biological Chemistry|June 22, 1999
Congenital insulin resistance associated with a conformational alteration in a conserved beta-sheet in the insulin receptor L1 domainM Rouard, J Bass, F Grigorescu, et al.
Diabetes|August 1, 1989
Polymorphic human insulin-responsive glucose-transporter gene on chromosome 17p13G I Bell, J C Murray, Y Nakamura, et al.
Molecular Endocrinology (Baltimore, Md.)|August 1, 1993
Human thyroxine-binding globulin gene: complete sequence and transcriptional regulationY Hayashi, Y Mori, O E Janssen, et al.
Biochemical and Biophysical Research Communications|September 30, 1985
Genes for insulin I and II, parathyroid hormone, and calcitonin are on rat chromosome 1S Todd, M C Yoshida, X E Fang, et al.
Diabetes|September 1, 1997
Mutations in the hepatocyte nuclear factor-1alpha/MODY3 gene in Japanese subjects with early- and late-onset NIDDMN Iwasaki, N Oda, M Ogata, et al.
Diabetes|January 1, 1994
Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigreeW H Herman, S S Fajans, F J Ortiz, et al.
Pageof 32

Showing results (241-250 of 317) with videos related to

Sort By:
Pageof 32
Diabetes|February 1, 1996
Identification of microsatellite markers near the human genes encoding the beta-cell ATP-sensitive K+ channel and linkage studies with NIDDM in JapaneseN Iwasaki, M Kawamura, K Yamagata, et al.
The Journal of Clinical Investigation|April 1, 1989
Sequence of the variant thyroxine-binding globulin of Australian aborigines. Only one of two amino acid replacements is responsible for its altered propertiesK Takeda, Y Mori, S Sobieszczyk, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 1, 1988
Sequence, tissue distribution, and chromosomal localization of mRNA encoding a human glucose transporter-like proteinH Fukumoto, S Seino, H Imura, et al.
Molecular Endocrinology (Baltimore, Md.)|December 1, 1992
Somatostatin receptors, an expanding gene family: cloning and functional characterization of human SSTR3, a protein coupled to adenylyl cyclaseY Yamada, T Reisine, S F Law, et al.
The Journal of Biological Chemistry|June 22, 1999
Congenital insulin resistance associated with a conformational alteration in a conserved beta-sheet in the insulin receptor L1 domainM Rouard, J Bass, F Grigorescu, et al.
Diabetes|August 1, 1989
Polymorphic human insulin-responsive glucose-transporter gene on chromosome 17p13G I Bell, J C Murray, Y Nakamura, et al.
Molecular Endocrinology (Baltimore, Md.)|August 1, 1993
Human thyroxine-binding globulin gene: complete sequence and transcriptional regulationY Hayashi, Y Mori, O E Janssen, et al.
Biochemical and Biophysical Research Communications|September 30, 1985
Genes for insulin I and II, parathyroid hormone, and calcitonin are on rat chromosome 1S Todd, M C Yoshida, X E Fang, et al.
Diabetes|September 1, 1997
Mutations in the hepatocyte nuclear factor-1alpha/MODY3 gene in Japanese subjects with early- and late-onset NIDDMN Iwasaki, N Oda, M Ogata, et al.
Diabetes|January 1, 1994
Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigreeW H Herman, S S Fajans, F J Ortiz, et al.
Pageof 32