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G J Walker

Showing results (81-90 of 92) with videos related to

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Melanoma Research|December 3, 2002
PTEN inactivation is rare in melanoma tumours but occurs frequently in melanoma cell linesP M Pollock, G J Walker, J M Glendening, et al.
Human Molecular Genetics|October 1, 1995
Mutations of the CDKN2/p16INK4 gene in Australian melanoma kindredsG J Walker, C J Hussussian, J F Flores, et al.
Experimental Lung Research|January 1, 1985
Delayed pulmonary maturation in the fetus of the streptozotocin-diabetic ratI H Gewolb, S A Rooney, C Barrett, et al.
American Journal of Human Genetics|July 6, 2000
Localization of multiple melanoma tumor-suppressor genes on chromosome 11 by use of homozygosity mapping-of-deletions analysisE K Goldberg, J M Glendening, Z Karanjawala, et al.
Oncogene|January 7, 1998
Analysis of the CDKN2A, CDKN2B and CDK4 genes in 48 Australian melanoma kindredsJ F Flores, P M Pollock, G J Walker, et al.
Cancer Research|December 11, 1997
Low frequency of p16/CDKN2A methylation in sporadic melanoma: comparative approaches for methylation analysis of primary tumorsM L Gonzalgo, C M Bender, E H You, et al.
American Journal of Human Genetics|October 1, 1993
Confirmation of chromosome 9p linkage in familial melanomaD J Nancarrow, G J Mann, E A Holland, et al.
Cancer Research|July 1, 1995
Loss of expression of the p16/cyclin-dependent kinase inhibitor 2 tumor suppressor gene in melanocytic lesions correlates with invasive stage of tumor progressionJ A Reed, F Loganzo, C R Shea, et al.
The Journal of Endocrinology|August 9, 2003
Activating point mutations in cyclin-dependent kinase 4 are not seen in sporadic pituitary adenomas, insulinomas or Leydig cell tumoursV V Vax, R Bibi, S Diaz-Cano, et al.
Oncogene|February 14, 2017
Melanocyte transformation requires complete loss of all pocket protein function via a mechanism that mitigates the need for MAPK pathway activationI D Tonks, P Mukhopadhyay, W A Schroder, et al.
Pageof 10

Showing results (81-90 of 92) with videos related to

Sort By:
Pageof 10
Melanoma Research|December 3, 2002
PTEN inactivation is rare in melanoma tumours but occurs frequently in melanoma cell linesP M Pollock, G J Walker, J M Glendening, et al.
Human Molecular Genetics|October 1, 1995
Mutations of the CDKN2/p16INK4 gene in Australian melanoma kindredsG J Walker, C J Hussussian, J F Flores, et al.
Experimental Lung Research|January 1, 1985
Delayed pulmonary maturation in the fetus of the streptozotocin-diabetic ratI H Gewolb, S A Rooney, C Barrett, et al.
American Journal of Human Genetics|July 6, 2000
Localization of multiple melanoma tumor-suppressor genes on chromosome 11 by use of homozygosity mapping-of-deletions analysisE K Goldberg, J M Glendening, Z Karanjawala, et al.
Oncogene|January 7, 1998
Analysis of the CDKN2A, CDKN2B and CDK4 genes in 48 Australian melanoma kindredsJ F Flores, P M Pollock, G J Walker, et al.
Cancer Research|December 11, 1997
Low frequency of p16/CDKN2A methylation in sporadic melanoma: comparative approaches for methylation analysis of primary tumorsM L Gonzalgo, C M Bender, E H You, et al.
American Journal of Human Genetics|October 1, 1993
Confirmation of chromosome 9p linkage in familial melanomaD J Nancarrow, G J Mann, E A Holland, et al.
Cancer Research|July 1, 1995
Loss of expression of the p16/cyclin-dependent kinase inhibitor 2 tumor suppressor gene in melanocytic lesions correlates with invasive stage of tumor progressionJ A Reed, F Loganzo, C R Shea, et al.
The Journal of Endocrinology|August 9, 2003
Activating point mutations in cyclin-dependent kinase 4 are not seen in sporadic pituitary adenomas, insulinomas or Leydig cell tumoursV V Vax, R Bibi, S Diaz-Cano, et al.
Oncogene|February 14, 2017
Melanocyte transformation requires complete loss of all pocket protein function via a mechanism that mitigates the need for MAPK pathway activationI D Tonks, P Mukhopadhyay, W A Schroder, et al.
Pageof 10