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Investigative Ophthalmology & Visual Science|April 14, 2017
Postretinal Structure and Function in Severe Congenital Photoreceptor Blindness Caused by Mutations in the GUCY2D GeneGeoffrey K Aguirre, Omar H Butt, Ritobrato Datta, et al.Investigative Ophthalmology & Visual Science|June 19, 2019
Treatment Potential for Macular Cone Vision in Leber Congenital Amaurosis Due to CEP290 or NPHP5 Mutations: Predictions From Artificial IntelligenceAlexander Sumaroka, Alexandra V Garafalo, Evelyn P Semenov, et al.Vision Research|October 6, 2001
Augmented rod bipolar cell function in partial receptor loss: an ERG study in P23H rhodopsin transgenic and aging normal ratsT S Aleman, M M LaVail, R Montemayor, et al.Nature Genetics|October 30, 2004
Lifespan and mitochondrial control of neurodegenerationAlan F Wright, Samuel G Jacobson, Artur V Cideciyan, et al.Investigative Ophthalmology & Visual Science|January 31, 2013
Abnormal thickening as well as thinning of the photoreceptor layer in intermediate age-related macular degenerationSam Sadigh, Artur V Cideciyan, Alexander Sumaroka, et al.Current Topics in Microbiology and Immunology|March 24, 1999
T helper differentiation proceeds through Stat1-dependent, Stat4-dependent and Stat4-independent phasesK M Murphy, W Ouyang, S J Szabo, et al.Annals of Surgery|September 1, 1988
Orthotopic transplantation during early infancy as therapy for incurable congenital heart diseaseL L Bailey, A N Assaad, R F Trimm, et al.Journal of the American Academy of Child and Adolescent Psychiatry|October 5, 2002
Fluoxetine for acute treatment of depression in children and adolescents: a placebo-controlled, randomized clinical trialGraham J Emslie, John H Heiligenstein, Karen Dineen Wagner, et al.Investigative Ophthalmology & Visual Science|February 20, 2014
Inner and outer retinal changes in retinal degenerations associated with ABCA4 mutationsWei Chieh Huang, Artur V Cideciyan, Alejandro J Roman, et al.American Journal of Human Genetics|October 27, 1997
Analysis of the RPGR gene in 11 pedigrees with the retinitis pigmentosa type 3 genotype: paucity of mutations in the coding region but splice defects in two familiesR Fujita, M Buraczynska, L Gieser, et al.Pageof 57