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G L Fry

Showing results (11-20 of 27) with videos related to

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Arteriosclerosis, Thrombosis, and Vascular Biology|November 10, 2001
Macrophage-specific expression of human lipoprotein lipase accelerates atherosclerosis in transgenic apolipoprotein e knockout mice but not in C57BL/6 miceK Wilson, G L Fry, D A Chappell, et al.
Cellular Immunology|April 15, 1984
Induction and amplification of T-lymphocyte proliferative responses to periodate and soybean agglutinin by human adult vascular endothelial cellsL Ercolani, T J Parsons, J C Hoak, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|August 18, 1981
Role of the vascular endotheliumJ C Hoak, R L Czervionke, G L Fry, et al.
The Journal of Clinical Investigation|May 1, 1979
Inhibition of prostacyclin by treatment of endothelium with aspirin. Correlation with platelet adherenceR L Czervionke, J B Smith, G L Fry, et al.
The Journal of Biological Chemistry|December 5, 1993
Low density lipoprotein receptors bind and mediate cellular catabolism of normal very low density lipoproteins in vitroD A Chappell, G L Fry, M A Waknitz, et al.
Transactions of the Association of American Physicians|January 1, 1982
Contribution of a platelet component to endothelial prostacyclin production despite inhibition of platelet cyclooxygenase activityR L Czervionke, J C Hoak, D L Haycraft, et al.
Progress in Clinical and Biological Research|January 1, 1982
Role of the vascular endotheliumJ C Hoak, W M Parks, G L Fry, et al.
Journal of Lipid Research|November 4, 2000
Lipoprotein lipase- and hepatic triglyceride lipase- promoted very low density lipoprotein degradation proceeds via an apolipoprotein E-dependent mechanismJ D Medh, G L Fry, S L Bowen, et al.
The Journal of Clinical Investigation|May 1, 1980
Effect of fatty acid modification on prostacyclin production by cultured human endothelial cellsA A Spector, J C Hoak, G L Fry, et al.
The Journal of Biological Chemistry|January 13, 1995
The 39-kDa receptor-associated protein modulates lipoprotein catabolism by binding to LDL receptorsJ D Medh, G L Fry, S L Bowen, et al.
Pageof 3

Showing results (11-20 of 27) with videos related to

Sort By:
Pageof 3
Arteriosclerosis, Thrombosis, and Vascular Biology|November 10, 2001
Macrophage-specific expression of human lipoprotein lipase accelerates atherosclerosis in transgenic apolipoprotein e knockout mice but not in C57BL/6 miceK Wilson, G L Fry, D A Chappell, et al.
Cellular Immunology|April 15, 1984
Induction and amplification of T-lymphocyte proliferative responses to periodate and soybean agglutinin by human adult vascular endothelial cellsL Ercolani, T J Parsons, J C Hoak, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|August 18, 1981
Role of the vascular endotheliumJ C Hoak, R L Czervionke, G L Fry, et al.
The Journal of Clinical Investigation|May 1, 1979
Inhibition of prostacyclin by treatment of endothelium with aspirin. Correlation with platelet adherenceR L Czervionke, J B Smith, G L Fry, et al.
The Journal of Biological Chemistry|December 5, 1993
Low density lipoprotein receptors bind and mediate cellular catabolism of normal very low density lipoproteins in vitroD A Chappell, G L Fry, M A Waknitz, et al.
Transactions of the Association of American Physicians|January 1, 1982
Contribution of a platelet component to endothelial prostacyclin production despite inhibition of platelet cyclooxygenase activityR L Czervionke, J C Hoak, D L Haycraft, et al.
Progress in Clinical and Biological Research|January 1, 1982
Role of the vascular endotheliumJ C Hoak, W M Parks, G L Fry, et al.
Journal of Lipid Research|November 4, 2000
Lipoprotein lipase- and hepatic triglyceride lipase- promoted very low density lipoprotein degradation proceeds via an apolipoprotein E-dependent mechanismJ D Medh, G L Fry, S L Bowen, et al.
The Journal of Clinical Investigation|May 1, 1980
Effect of fatty acid modification on prostacyclin production by cultured human endothelial cellsA A Spector, J C Hoak, G L Fry, et al.
The Journal of Biological Chemistry|January 13, 1995
The 39-kDa receptor-associated protein modulates lipoprotein catabolism by binding to LDL receptorsJ D Medh, G L Fry, S L Bowen, et al.
Pageof 3