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G M Omann

Showing results (41-50 of 50) with videos related to

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Brain Research|June 25, 1993
A cellular model of oxidant-mediated neuronal injuryD B Hinshaw, M T Miller, G M Omann, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 15, 1992
A threshold level of coupled G-proteins is required to transduce neutrophil responsesG M Omann, J M Harter, N Hassan, et al.
Journal of Leukocyte Biology|November 1, 1986
Transient catecholamine modulation of neutrophil activation: kinetic and intracellular aspects of isoproterenol actionE S Tecoma, H J Motulsky, A E Traynor, et al.
Archives of Biochemistry and Biophysics|May 23, 2001
Inhibition of mono-ADP-ribosyltransferase activity during the execution phase of apoptosis prevents apoptotic body formationI J Lodhi, R E Clift, G M Omann, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 15, 1987
N-formylpeptide-receptor dynamics, cytoskeletal activation, and intracellular calcium response in human neutrophil cytoplastsG M Omann, W N Swann, Z G Oades, et al.
Physiological Reviews|January 1, 1987
Signal transduction and cytoskeletal activation in the neutrophilG M Omann, R A Allen, G M Bokoch, et al.
The Journal of Biological Chemistry|September 15, 1989
Transient increase in phosphatidylinositol 3,4-bisphosphate and phosphatidylinositol trisphosphate during activation of human neutrophilsA E Traynor-Kaplan, B L Thompson, A L Harris, et al.
The American Journal of Physiology|May 1, 1993
ATP depletion induces an increase in the assembly of a labile pool of polymerized actin in endothelial cellsD B Hinshaw, J M Burger, M T Miller, et al.
The Journal of Biological Chemistry|September 25, 1985
Signal transduction and ligand-receptor dynamics in the human neutrophil. Transient responses and occupancy-response relations at the formyl peptide receptorL A Sklar, P A Hyslop, Z G Oades, et al.
Blood|February 9, 2000
Actin cytoskeletal function is spared, but apoptosis is increased, in WAS patient hematopoietic cellsR Rengan, H D Ochs, L I Sweet, et al.
Pageof 5

Showing results (41-50 of 50) with videos related to

Sort By:
Pageof 5
You have reached the last page of results.This site can display upto 50 results.
Brain Research|June 25, 1993
A cellular model of oxidant-mediated neuronal injuryD B Hinshaw, M T Miller, G M Omann, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 15, 1992
A threshold level of coupled G-proteins is required to transduce neutrophil responsesG M Omann, J M Harter, N Hassan, et al.
Journal of Leukocyte Biology|November 1, 1986
Transient catecholamine modulation of neutrophil activation: kinetic and intracellular aspects of isoproterenol actionE S Tecoma, H J Motulsky, A E Traynor, et al.
Archives of Biochemistry and Biophysics|May 23, 2001
Inhibition of mono-ADP-ribosyltransferase activity during the execution phase of apoptosis prevents apoptotic body formationI J Lodhi, R E Clift, G M Omann, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 15, 1987
N-formylpeptide-receptor dynamics, cytoskeletal activation, and intracellular calcium response in human neutrophil cytoplastsG M Omann, W N Swann, Z G Oades, et al.
Physiological Reviews|January 1, 1987
Signal transduction and cytoskeletal activation in the neutrophilG M Omann, R A Allen, G M Bokoch, et al.
The Journal of Biological Chemistry|September 15, 1989
Transient increase in phosphatidylinositol 3,4-bisphosphate and phosphatidylinositol trisphosphate during activation of human neutrophilsA E Traynor-Kaplan, B L Thompson, A L Harris, et al.
The American Journal of Physiology|May 1, 1993
ATP depletion induces an increase in the assembly of a labile pool of polymerized actin in endothelial cellsD B Hinshaw, J M Burger, M T Miller, et al.
The Journal of Biological Chemistry|September 25, 1985
Signal transduction and ligand-receptor dynamics in the human neutrophil. Transient responses and occupancy-response relations at the formyl peptide receptorL A Sklar, P A Hyslop, Z G Oades, et al.
Blood|February 9, 2000
Actin cytoskeletal function is spared, but apoptosis is increased, in WAS patient hematopoietic cellsR Rengan, H D Ochs, L I Sweet, et al.
Pageof 5