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G M Pasinetti

Showing results (51-60 of 62) with videos related to

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Archives of Neurology|August 6, 2000
Cytokine gene expression as a function of the clinical progression of Alzheimer disease dementiaJ D Luterman, V Haroutunian, S Yemul, et al.
The American Journal of Pathology|September 17, 1999
Potentiation of excitotoxicity in transgenic mice overexpressing neuronal cyclooxygenase-2K A Kelley, L Ho, D Winger, et al.
Pharmacological Research|May 1, 1989
Combined in situ hybridization and immunocytochemistry in the assay of pharmacological effects on tyrosine hydroxylase mRNA concentrationG M Pasinetti, D G Morgan, S A Johnson, et al.
Neuroscience|October 1, 1994
Selective expression of clusterin (SGP-2) and complement C1qB and C4 during responses to neurotoxins in vivo and in vitroI Rozovsky, T E Morgan, D A Willoughby, et al.
Neurobiology of Disease|June 1, 1996
Hereditary deficiencies in complement C5 are associated with intensified neurodegenerative responses that implicate new roles for the C-system in neuronal and astrocytic functionsG M Pasinetti, G Tocco, S Sakhi, et al.
Cardiovascular Psychiatry and Neurology|November 26, 2009
Role of Hypertension in Aggravating Abeta Neuropathology of AD Type and Tau-Mediated Motor ImpairmentC Díaz-Ruiz, J Wang, H Ksiezak-Reding, et al.
Brain Research. Molecular Brain Research|March 11, 1992
Slow changes of tyrosine hydroxylase gene expression in dopaminergic brain neurons after neurotoxin lesioning: a model for neuron agingG M Pasinetti, H H Osterburg, A B Kelly, et al.
Neuroscience Letters|February 13, 2001
Altered expression of a-type but not b-type synapsin isoform in the brain of patients at high risk for Alzheimer's disease assessed by DNA microarray techniqueL Ho, Y Guo, L Spielman, et al.
Experimental Neurology|November 1, 1992
Complement C1qB and C4 mRNAs responses to lesioning in rat brainG M Pasinetti, S A Johnson, I Rozovsky, et al.
Archives of Neurology|March 20, 2001
Neuronal cyclooxygenase 2 expression in the hippocampal formation as a function of the clinical progression of Alzheimer diseaseL Ho, D Purohit, V Haroutunian, et al.
Pageof 7

Showing results (51-60 of 62) with videos related to

Sort By:
Pageof 7
Archives of Neurology|August 6, 2000
Cytokine gene expression as a function of the clinical progression of Alzheimer disease dementiaJ D Luterman, V Haroutunian, S Yemul, et al.
The American Journal of Pathology|September 17, 1999
Potentiation of excitotoxicity in transgenic mice overexpressing neuronal cyclooxygenase-2K A Kelley, L Ho, D Winger, et al.
Pharmacological Research|May 1, 1989
Combined in situ hybridization and immunocytochemistry in the assay of pharmacological effects on tyrosine hydroxylase mRNA concentrationG M Pasinetti, D G Morgan, S A Johnson, et al.
Neuroscience|October 1, 1994
Selective expression of clusterin (SGP-2) and complement C1qB and C4 during responses to neurotoxins in vivo and in vitroI Rozovsky, T E Morgan, D A Willoughby, et al.
Neurobiology of Disease|June 1, 1996
Hereditary deficiencies in complement C5 are associated with intensified neurodegenerative responses that implicate new roles for the C-system in neuronal and astrocytic functionsG M Pasinetti, G Tocco, S Sakhi, et al.
Cardiovascular Psychiatry and Neurology|November 26, 2009
Role of Hypertension in Aggravating Abeta Neuropathology of AD Type and Tau-Mediated Motor ImpairmentC Díaz-Ruiz, J Wang, H Ksiezak-Reding, et al.
Brain Research. Molecular Brain Research|March 11, 1992
Slow changes of tyrosine hydroxylase gene expression in dopaminergic brain neurons after neurotoxin lesioning: a model for neuron agingG M Pasinetti, H H Osterburg, A B Kelly, et al.
Neuroscience Letters|February 13, 2001
Altered expression of a-type but not b-type synapsin isoform in the brain of patients at high risk for Alzheimer's disease assessed by DNA microarray techniqueL Ho, Y Guo, L Spielman, et al.
Experimental Neurology|November 1, 1992
Complement C1qB and C4 mRNAs responses to lesioning in rat brainG M Pasinetti, S A Johnson, I Rozovsky, et al.
Archives of Neurology|March 20, 2001
Neuronal cyclooxygenase 2 expression in the hippocampal formation as a function of the clinical progression of Alzheimer diseaseL Ho, D Purohit, V Haroutunian, et al.
Pageof 7