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The American Journal of Physiology|April 1, 1990
Arachidonic acid causes postischemic dysfunction in control but not diabetic heartsG M PieperCardiovascular Research|December 1, 1988
Superoxide dismutase plus catalase improves post-ischaemic recovery in the diabetic heartG M PieperLife Sciences|January 1, 1997
Divergent actions of chronic insulin treatment in vivo versus acute treatment ex vivo on diabetic-induced endothelial dysfunctionG M PieperJournal of Cardiovascular Pharmacology|January 1, 1997
Acute amelioration of diabetic endothelial dysfunction with a derivative of the nitric oxide synthase cofactor, tetrahydrobiopterinG M PieperJournal of Cardiovascular Pharmacology|January 22, 1998
Diabetes-induced endothelial dysfunction is prevented by long-term treatment with the modified iron chelator, hydroxyethyl starch conjugated-deferoxamineG M Pieper, W SiebeneichEuropean Journal of Pharmacology|September 2, 2000
Temocapril, an angiotensin converting enzyme inhibitor, protects against diabetes-induced endothelial dysfunctionG M Pieper, W SiebeneichCardiovascular Research|April 1, 1997
Glucose elevations alter bradykinin-stimulated intracellular calcium accumulation in cultured endothelial cellsG M Pieper, L DondlingerThe Journal of Pharmacology and Experimental Therapeutics|October 23, 1997
Use of a nitronyl nitroxide to discriminate the contribution of nitric oxide radical in endothelium-dependent relaxation of control and diabetic blood vesselsG M Pieper, W SiebeneichThe American Journal of Physiology|July 1, 1992
Regulation of spontaneous EDRF release in diabetic rat aorta by oxygen free radicalsP Langenstroer, G M PieperPageof 9