G M Wray - JoVE Visualize

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Shock (Augusta, Ga.)|August 4, 1998

Effects of inhibitors of poly(ADP-ribose) synthetase activity on hypotension and multiple organ dysfunction caused by endotoxinG M Wray, C J Hinds, C Thiemermann

FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 4, 1999

Identification of copper/zinc superoxide dismutase as a novel nitric oxide-regulated gene in rat glomerular mesangial cells and kidneys of endotoxemic ratsS Frank, K Zacharowski, G M Wray, et al.

Shock (Augusta, Ga.)|June 9, 1998

Selective inhibition of the activity of inducible nitric oxide synthase prevents the circulatory failure, but not the organ injury/dysfunction, caused by endotoxinG M Wray, C G Millar, C J Hinds, et al.

Shock (Augusta, Ga.)|February 28, 2001

A cell wall component from pathogenic and non-pathogenic gram-positive bacteria (peptidoglycan) synergises with endotoxin to cause the release of tumour necrosis factor-alpha, nitric oxide production, shock, and multiple organ injury/dysfunction in the ratG M Wray, S J Foster, C J Hinds, et al.

British Journal of Pharmacology|July 1, 1998

Effects of inhibitors of the activity of cyclo-oxygenase-2 on the hypotension and multiple organ dysfunction caused by endotoxin: a comparison with dexamethasoneM Leach, L C Hamilton, A Olbrich, et al.

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Showing results (1-10 of 5) with videos related to

Sort By:

Pageof 1

Shock (Augusta, Ga.)|August 4, 1998

Effects of inhibitors of poly(ADP-ribose) synthetase activity on hypotension and multiple organ dysfunction caused by endotoxinG M Wray, C J Hinds, C Thiemermann

FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 4, 1999

Identification of copper/zinc superoxide dismutase as a novel nitric oxide-regulated gene in rat glomerular mesangial cells and kidneys of endotoxemic ratsS Frank, K Zacharowski, G M Wray, et al.

Shock (Augusta, Ga.)|June 9, 1998

Selective inhibition of the activity of inducible nitric oxide synthase prevents the circulatory failure, but not the organ injury/dysfunction, caused by endotoxinG M Wray, C G Millar, C J Hinds, et al.

Shock (Augusta, Ga.)|February 28, 2001

A cell wall component from pathogenic and non-pathogenic gram-positive bacteria (peptidoglycan) synergises with endotoxin to cause the release of tumour necrosis factor-alpha, nitric oxide production, shock, and multiple organ injury/dysfunction in the ratG M Wray, S J Foster, C J Hinds, et al.

British Journal of Pharmacology|July 1, 1998

Effects of inhibitors of the activity of cyclo-oxygenase-2 on the hypotension and multiple organ dysfunction caused by endotoxin: a comparison with dexamethasoneM Leach, L C Hamilton, A Olbrich, et al.

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