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The Biochemical Journal|June 15, 1989
Guanine-nucleotide-binding proteins Gi and Gs in fat-cells from normal, hypothyroid and obese human subjectsJ J Ohisalo, G MilliganTrends in Pharmacological Sciences|February 12, 1998
Inverse agonism and the regulation of receptor numberG Milligan, R A BondCellular Signalling|January 1, 1990
Biphasic regulation of adenylate cyclase by cholera toxin in neuroblastoma x glioma hybrid cells is due to the activation and subsequent loss of the alpha subunit of the stimulatory GTP binding protein (GS)K G Macleod, G MilliganMolecular Pharmacology|July 1, 1994
The gonadotrophin-releasing hormone receptor of alpha T3-1 pituitary cells regulates cellular levels of both of the phosphoinositidase C-linked G proteins, Gq alpha and G11 alpha, equallyB H Shah, G MilliganBiochimica Et Biophysica Acta|July 21, 1994
Concurrent specific immunological detection of both primate and rodent forms of the guanine nucleotide binding protein G11 alpha following their coexpressionG D Kim, G MilliganBiochemical and Biophysical Research Communications|May 16, 1994
Regulation of p42 and p44 MAP kinase isoforms in Rat-1 fibroblasts stably transfected with alpha 2C10 adrenoreceptorsN G Anderson, G MilliganThe Biochemical Journal|June 15, 1994
Agonist regulation of cellular Gs alpha-subunit levels in neuroblastoma x glioma hybrid NG108-15 cells transfected to express different levels of the human beta 2 adrenoceptorE J Adie, G MilliganCellular Signalling|July 1, 1993
An arginine residue is the site of receptor-stimulated, cholera toxin-catalysed ADP-ribosylation of pertussis toxin-sensitive G-proteinsG Milligan, F M MitchellThe Biochemical Journal|April 1, 1991
Cholera toxin impairment of opioid-mediated inhibition of adenylate cyclase in neuroblastoma x glioma hybrid cells is due to a toxin-induced decrease in opioid receptor levelsF R McKenzie, G MilliganNeurochemistry International|May 27, 2010
The effect of tunicamycin on prostacyclin (PGI(2)) receptors of neuronal hybrid cellsJ M Hall, M Borland, F W Hemming, et al.Pageof 39