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The Journal of Biological Chemistry|March 25, 1990
Down-regulation of Gi sub-types by prolonged incubation of adipocytes with an A1 adenosine receptor agonistA Green, J L Johnson, G MilliganThe Journal of Biological Chemistry|February 15, 1992
Gi down-regulation as a mechanism for heterologous desensitization in adipocytesA Green, G Milligan, S B DobiasThe Biochemical Journal|January 15, 1988
Treatment of streptozotocin-diabetic rats with metformin restores the ability of insulin to inhibit adenylate cyclase activity and demonstrates that insulin does not exert this action through the inhibitory guanine nucleotide regulatory protein GiD Gawler, G Milligan, M D HouslayTrends in Biochemical Sciences|May 1, 1995
The dynamic role of palmitoylation in signal transductionG Milligan, M Parenti, A I MageeThe Biochemical Journal|July 1, 1997
Measurement of agonist-induced guanine nucleotide turnover by the G-protein Gi1alpha when constrained within an alpha2A-adrenoceptor-Gi1alpha fusion proteinA Wise, I C Carr, G MilliganThe Biochemical Journal|April 1, 1992
Regulation of cardiac adenylate cyclase activity in rodent models of obesityD Strassheim, M D Houslay, G MilliganThe Journal of Biological Chemistry|January 8, 2000
The G protein alpha subunit has a key role in determining the specificity of coupling to, but not the activation of, G protein-gated inwardly rectifying K(+) channelsJ L Leaney, G Milligan, A TinkerThe Journal of Biological Chemistry|May 16, 2000
The regulator of G protein signaling RGS4 selectively enhances alpha 2A-adreoreceptor stimulation of the GTPase activity of Go1alpha and Gi2alphaA Cavalli, K M Druey, G MilliganThe Biochemical Journal|March 1, 1990
Diabetes abolishes the GTP-dependent, but not the receptor-dependent inhibitory function of the inhibitory guanine-nucleotide-binding regulatory protein (Gi) on adipocyte adenylate cyclase activityD Strassheim, G Milligan, M D HouslayThe Journal of Endocrinology|January 1, 1993
Characterization of the gonadotrophin-releasing hormone receptor in alpha T3-1 pituitary gonadotroph cellsL Anderson, G Milligan, K A EidnePageof 39