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G Milligan

Showing results (71-80 of 356) with videos related to

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The Biochemical Journal|September 15, 1990
Concurrent up-regulation of guanine-nucleotide-binding proteins Gi1 alpha, Gi2 alpha and Gi3 alpha in adipocytes of hypothyroid ratsG Milligan, E D Saggerson
British Journal of Pharmacology|September 2, 2009
GPCR theme editorialG Milligan, J C McGrath
British Journal of Pharmacology|March 7, 1998
Efficacy of inverse agonists in cells overexpressing a constitutively active beta2-adrenoceptor and type II adenylyl cyclaseP A Stevens, G Milligan
Progress in Brain Research|January 1, 1982
The use of biochemical methods for estimating membrane potentialG Milligan, P G Strange
FEBS Letters|January 6, 2000
An Asp79Asn mutation of the alpha2A-adrenoceptor interferes equally with agonist activation of individual Gialpha-family G protein subtypesR J Ward, G Milligan
The Biochemical Journal|November 1, 1994
Regulation of basal adenylate cyclase activity in neuroblastoma x glioma hybrid, NG108-15, cells transfected to express the human beta 2 adrenoceptor: evidence for empty receptor stimulation of the adenylate cyclase cascadeE J Adie, G Milligan
Molecular Ecology|April 1, 1994
Analysis of population genetic structure with RAPD markersM Lynch, B G Milligan
The Biochemical Journal|June 15, 1989
Guanine-nucleotide-binding proteins Gi and Gs in fat-cells from normal, hypothyroid and obese human subjectsJ J Ohisalo, G Milligan
Trends in Pharmacological Sciences|February 12, 1998
Inverse agonism and the regulation of receptor numberG Milligan, R A Bond
Cellular Signalling|January 1, 1990
Biphasic regulation of adenylate cyclase by cholera toxin in neuroblastoma x glioma hybrid cells is due to the activation and subsequent loss of the alpha subunit of the stimulatory GTP binding protein (GS)K G Macleod, G Milligan
Pageof 36

Showing results (71-80 of 356) with videos related to

Sort By:
Pageof 36
The Biochemical Journal|September 15, 1990
Concurrent up-regulation of guanine-nucleotide-binding proteins Gi1 alpha, Gi2 alpha and Gi3 alpha in adipocytes of hypothyroid ratsG Milligan, E D Saggerson
British Journal of Pharmacology|September 2, 2009
GPCR theme editorialG Milligan, J C McGrath
British Journal of Pharmacology|March 7, 1998
Efficacy of inverse agonists in cells overexpressing a constitutively active beta2-adrenoceptor and type II adenylyl cyclaseP A Stevens, G Milligan
Progress in Brain Research|January 1, 1982
The use of biochemical methods for estimating membrane potentialG Milligan, P G Strange
FEBS Letters|January 6, 2000
An Asp79Asn mutation of the alpha2A-adrenoceptor interferes equally with agonist activation of individual Gialpha-family G protein subtypesR J Ward, G Milligan
The Biochemical Journal|November 1, 1994
Regulation of basal adenylate cyclase activity in neuroblastoma x glioma hybrid, NG108-15, cells transfected to express the human beta 2 adrenoceptor: evidence for empty receptor stimulation of the adenylate cyclase cascadeE J Adie, G Milligan
Molecular Ecology|April 1, 1994
Analysis of population genetic structure with RAPD markersM Lynch, B G Milligan
The Biochemical Journal|June 15, 1989
Guanine-nucleotide-binding proteins Gi and Gs in fat-cells from normal, hypothyroid and obese human subjectsJ J Ohisalo, G Milligan
Trends in Pharmacological Sciences|February 12, 1998
Inverse agonism and the regulation of receptor numberG Milligan, R A Bond
Cellular Signalling|January 1, 1990
Biphasic regulation of adenylate cyclase by cholera toxin in neuroblastoma x glioma hybrid cells is due to the activation and subsequent loss of the alpha subunit of the stimulatory GTP binding protein (GS)K G Macleod, G Milligan
Pageof 36