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G Mohapatra

Showing results (11-20 of 31) with videos related to

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The American Journal of Pathology|January 1, 1997
Gain of chromosome 17 is the most frequent abnormality detected in neuroblastoma by comparative genomic hybridizationD Plantaz, G Mohapatra, K K Matthay, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 2, 1999
Chromosomal abnormalities in glioblastoma multiforme by comparative genomic hybridization: correlation with radiation treatment outcomeS L Huhn, G Mohapatra, A Bollen, et al.
Oncogene|March 7, 2000
HYAL1LUCA-1, a candidate tumor suppressor gene on chromosome 3p21.3, is inactivated in head and neck squamous cell carcinomas by aberrant splicing of pre-mRNAG I Frost, G Mohapatra, T M Wong, et al.
Cancer Genetics and Cytogenetics|September 1, 1995
Heterogeneity, polyploidy, aneusomy, and 9p deletion in human glioblastoma multiformeS H Park, T Maeda, G Mohapatra, et al.
Genomics|October 27, 1997
FISH probes for mouse chromosome identificationY P Shi, G Mohapatra, J Miller, et al.
Cancer Genetics and Cytogenetics|December 24, 1997
Gliomas in families: chromosomal analysis by comparative genomic hybridizationA Patel, D J van Meyel, G Mohapatra, et al.
Genes, Chromosomes & Cancer|April 2, 1998
Genetic analysis of glioblastoma multiforme provides evidence for subgroups within the gradeG Mohapatra, A W Bollen, D H Kim, et al.
Genomics|March 21, 1998
The hyaluronidase gene HYAL1 maps to chromosome 3p21.2-p21.3 in human and 9F1-F2 in mouse, a conserved candidate tumor suppressor locusA B Csóka, G I Frost, H H Heng, et al.
Cancer|July 27, 1999
Comparative genomic hybridization in patients with supratentorial and infratentorial primitive neuroectodermal tumorsC Russo, M Pellarin, O Tingby, et al.
Journal of the Egyptian National Cancer Institute|January 29, 2024
PD-L1 expression and its significance in advanced NSCLC: real-world experience from a tertiary care centerSindhu Kilaru, Soumya Surath Panda, Lalatendu Moharana, et al.
Pageof 4

Showing results (11-20 of 31) with videos related to

Sort By:
Pageof 4
The American Journal of Pathology|January 1, 1997
Gain of chromosome 17 is the most frequent abnormality detected in neuroblastoma by comparative genomic hybridizationD Plantaz, G Mohapatra, K K Matthay, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 2, 1999
Chromosomal abnormalities in glioblastoma multiforme by comparative genomic hybridization: correlation with radiation treatment outcomeS L Huhn, G Mohapatra, A Bollen, et al.
Oncogene|March 7, 2000
HYAL1LUCA-1, a candidate tumor suppressor gene on chromosome 3p21.3, is inactivated in head and neck squamous cell carcinomas by aberrant splicing of pre-mRNAG I Frost, G Mohapatra, T M Wong, et al.
Cancer Genetics and Cytogenetics|September 1, 1995
Heterogeneity, polyploidy, aneusomy, and 9p deletion in human glioblastoma multiformeS H Park, T Maeda, G Mohapatra, et al.
Genomics|October 27, 1997
FISH probes for mouse chromosome identificationY P Shi, G Mohapatra, J Miller, et al.
Cancer Genetics and Cytogenetics|December 24, 1997
Gliomas in families: chromosomal analysis by comparative genomic hybridizationA Patel, D J van Meyel, G Mohapatra, et al.
Genes, Chromosomes & Cancer|April 2, 1998
Genetic analysis of glioblastoma multiforme provides evidence for subgroups within the gradeG Mohapatra, A W Bollen, D H Kim, et al.
Genomics|March 21, 1998
The hyaluronidase gene HYAL1 maps to chromosome 3p21.2-p21.3 in human and 9F1-F2 in mouse, a conserved candidate tumor suppressor locusA B Csóka, G I Frost, H H Heng, et al.
Cancer|July 27, 1999
Comparative genomic hybridization in patients with supratentorial and infratentorial primitive neuroectodermal tumorsC Russo, M Pellarin, O Tingby, et al.
Journal of the Egyptian National Cancer Institute|January 29, 2024
PD-L1 expression and its significance in advanced NSCLC: real-world experience from a tertiary care centerSindhu Kilaru, Soumya Surath Panda, Lalatendu Moharana, et al.
Pageof 4