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Physiological Chemistry and Physics and Medical NMR|January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part I. The endogenous mechanism of self-destruction within the malignant cell, and the roles of endotoxin, certain hormones and drugs, and active oxygen in causing cellular injury and deathG R JonesJournal of General Microbiology|July 1, 1975
A comparison of analytical methods for the numerical taxonomy of yeastsG R JonesMedical Hypotheses|January 3, 2001
Causes of Alzheimer's disease: paracetamol (acetaminophen) today? Amphetamines tomorrow?G R JonesPhysiological Chemistry and Physics and Medical NMR|January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part III. Spontaneous drug resistance, selectivity of antineoplastic action, and strategies for intensifying tumor injuryG R JonesFree Radical Research Communications|January 1, 1987
Dysbaric osteonecrosis (caisson disease of bone): are active oxygen species and the endocrine system responsible, and can control of the production of free radicals and their reaction products confer protection?G R JonesMedical Hypotheses|March 1, 1976
Cancer: restriction of chemical energy by fatty acids as the common pathway whereby various anti-tumour procedures selectively damage malignant cells in situG R JonesOncology|January 1, 1983
Interference with energy production as a means of causing injury to a human pancreatic carcinomaG R JonesPhysiological Chemistry and Physics and Medical NMR|January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part II. Lipid peroxidation and cell death; drug resistance as a consequence of reversible cellular injuryG R JonesPhysiological Chemistry and Physics and Medical NMR|January 1, 1991
Cancer therapy at the crossroads: which direction for the future?G R JonesPageof 19