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Neuroscience Letters|April 9, 1981
Adenosine depression of hippocampal neurons in vitro: an intracellular study of dose-dependent actions on synaptic and membrane potentialsG R Siggins, P SchubertProceedings of the National Academy of Sciences of the United States of America|August 1, 1981
Morphine and opioid peptides reduce inhibitory synaptic potentials in hippocampal pyramidal cells in vitro without alteration of membrane potentialG R Siggins, W ZieglgänsbergerJournal of Neurophysiology|April 14, 1999
Somatostatin acts in CA1 and CA3 to reduce hippocampal epileptiform activityM K Tallent, G R SigginsProceedings of the National Academy of Sciences of the United States of America|November 1, 1979
Chronic treatment with lithium or desipramine alters discharge frequency and norepinephrine responsiveness of cerebellar Purkinje cellsG R Siggins, J E SchultzScience (New York, N.Y.)|August 15, 1975
Analogs of cyclic adenosine monophosphate: correlation of inhibition of Purkinje Neurons with Protein Kinase ActivationG R Siggins, S J HenriksenJournal of Neurophysiology|February 7, 1998
Somatostatin depresses excitatory but not inhibitory neurotransmission in rat CA1 hippocampusM K Tallent, G R SigginsBrain Research|September 28, 1981
Somatostatin hyperpolarizes hippocampal pyramidal cells in vitroQ J Pittman, G R SigginsRegulatory Peptides|October 1, 1980
An iontophoretic survey of opioid peptide actions in the rat limbic system: in search of opiate epileptogenic mechanismsE D French, G R SigginsThe Journal of Pharmacology and Experimental Therapeutics|December 23, 1998
Chronic morphine treatment selectively augments metabotropic glutamate receptor-induced inhibition of N-methyl-D-aspartate receptor-mediated neurotransmission in nucleus accumbensG Martin, R Przewlocki, G R SigginsProceedings of the National Academy of Sciences of the United States of America|May 24, 2000
Dynamic actin filaments are required for stable long-term potentiation (LTP) in area CA1 of the hippocampusT Krucker, G R Siggins, S HalpainPageof 35