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G R Stark

Showing results (181-190 of 197) with videos related to

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Nucleic Acids Research|February 11, 1991
Differential response of the human 6-16 and 9-27 genes to alpha and gamma interferonsA M Ackrill, L E Reid, C S Gilbert, et al.
Nature|November 11, 1993
Complementation by the protein tyrosine kinase JAK2 of a mutant cell line defective in the interferon-gamma signal transduction pathwayD Watling, D Guschin, M Müller, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|January 30, 1995
Regulation and mechanisms of gene amplificationK A Smith, M L Agarwal, M V Chernov, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 17, 1996
Use of genetic suppressor elements to dissect distinct biological effects of separate p53 domainsV S Ossovskaya, I A Mazo, M V Chernov, et al.
The EMBO Journal|October 16, 1995
Mutant U5A cells are complemented by an interferon-alpha beta receptor subunit generated by alternative processing of a new member of a cytokine receptor gene clusterG Lutfalla, S J Holland, E Cinato, et al.
Immunity|November 1, 1994
Molecular analysis of G1B and G3A IFN gamma mutants reveals that defects in CIITA or RFX result in defective class II MHC and Ii gene inductionK C Chin, C Mao, C Skinner, et al.
The EMBO Journal|February 15, 1996
Kinase-negative mutants of JAK1 can sustain interferon-gamma-inducible gene expression but not an antiviral stateJ Briscoe, N C Rogers, B A Witthuhn, et al.
The Journal of Biological Chemistry|June 23, 1998
Growth hormone and prolactin stimulate tyrosine phosphorylation of insulin receptor substrate-1, -2, and -3, their association with p85 phosphatidylinositol 3-kinase (PI3-kinase), and concomitantly PI3-kinase activation via JAK2 kinaseT Yamauchi, Y Kaburagi, K Ueki, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 6, 1995
Combinatorial association and abundance of components of interferon-stimulated gene factor 3 dictate the selectivity of interferon responsesH A Bluyssen, R Muzaffar, R J Vlieststra, et al.
Journal of Virology|October 1, 1983
Persistent infection of some standard cell lines by lymphocytic choriomeningitis virus: transmission of infection by an intracellular agentB A van der Zeijst, B E Noyes, M E Mirault, et al.
Pageof 20

Showing results (181-190 of 197) with videos related to

Sort By:
Pageof 20
Nucleic Acids Research|February 11, 1991
Differential response of the human 6-16 and 9-27 genes to alpha and gamma interferonsA M Ackrill, L E Reid, C S Gilbert, et al.
Nature|November 11, 1993
Complementation by the protein tyrosine kinase JAK2 of a mutant cell line defective in the interferon-gamma signal transduction pathwayD Watling, D Guschin, M Müller, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|January 30, 1995
Regulation and mechanisms of gene amplificationK A Smith, M L Agarwal, M V Chernov, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 17, 1996
Use of genetic suppressor elements to dissect distinct biological effects of separate p53 domainsV S Ossovskaya, I A Mazo, M V Chernov, et al.
The EMBO Journal|October 16, 1995
Mutant U5A cells are complemented by an interferon-alpha beta receptor subunit generated by alternative processing of a new member of a cytokine receptor gene clusterG Lutfalla, S J Holland, E Cinato, et al.
Immunity|November 1, 1994
Molecular analysis of G1B and G3A IFN gamma mutants reveals that defects in CIITA or RFX result in defective class II MHC and Ii gene inductionK C Chin, C Mao, C Skinner, et al.
The EMBO Journal|February 15, 1996
Kinase-negative mutants of JAK1 can sustain interferon-gamma-inducible gene expression but not an antiviral stateJ Briscoe, N C Rogers, B A Witthuhn, et al.
The Journal of Biological Chemistry|June 23, 1998
Growth hormone and prolactin stimulate tyrosine phosphorylation of insulin receptor substrate-1, -2, and -3, their association with p85 phosphatidylinositol 3-kinase (PI3-kinase), and concomitantly PI3-kinase activation via JAK2 kinaseT Yamauchi, Y Kaburagi, K Ueki, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 6, 1995
Combinatorial association and abundance of components of interferon-stimulated gene factor 3 dictate the selectivity of interferon responsesH A Bluyssen, R Muzaffar, R J Vlieststra, et al.
Journal of Virology|October 1, 1983
Persistent infection of some standard cell lines by lymphocytic choriomeningitis virus: transmission of infection by an intracellular agentB A van der Zeijst, B E Noyes, M E Mirault, et al.
Pageof 20