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Pharmacogenetics
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February 18, 1999
An additional defective allele, CYP2C19*5, contributes to the S-mephenytoin poor metabolizer phenotype in Caucasians
G C Ibeanu, J Blaisdell, B I Ghanayem, et al.
Urologic Oncology
|
January 14, 2011
Environmental procarcinogen hypothesis of bladder cancer in humans: Dapsone hydroxylation as a susceptibility risk factor for aggressive bladder cancer
R Persad, C Fleming, H D Chern, et al.
Clinical Pharmacology and Therapeutics
|
September 1, 1990
Scleroderma is associated with differences in individual routes of drug metabolism: a study with dapsone, debrisoquin, and mephenytoin
D G May, C M Black, N J Olsen, et al.
Pharmacogenetics
|
January 1, 1995
The procarcinogen hypothesis for bladder cancer: activities of individual drug metabolizing enzymes as risk factors
R A Branch, H D Chern, A Adedoyin, et al.
Clinical Pharmacology and Therapeutics
|
August 1, 1986
Encainide disposition in patients with chronic cirrhosis
R H Bergstrand, T Wang, D M Roden, et al.
The Journal of Pharmacology and Experimental Therapeutics
|
January 22, 1998
A new genetic defect in human CYP2C19: mutation of the initiation codon is responsible for poor metabolism of S-mephenytoin
R J Ferguson, S M De Morais, S Benhamou, et al.
Pharmaceutical Research
|
April 23, 1999
Interrelationship between substrates and inhibitors of human CYP3A and P-glycoprotein
R B Kim, C Wandel, B Leake, et al.
Pharmacogenetics
|
December 12, 2001
In-vivo phenotyping for CYP3A by a single-point determination of midazolam plasma concentration
Y S Lin, G F Lockwood, M A Graham, et al.
Clinical Pharmacology and Therapeutics
|
August 15, 2001
Identification of functionally variant MDR1 alleles among European Americans and African Americans
R B Kim, B F Leake, E F Choo, et al.
AIDS Research and Human Retroviruses
|
October 29, 2000
Evidence of a source of HIV type 1 within the central nervous system by ultraintensive sampling of cerebrospinal fluid and plasma
D W Haas, L A Clough, B W Johnson, et al.
Page
of 19
Search research articles
Search
Showing results (181-190 of 190) with videos related to
Sort By:
Page
of 19
You have reached the last page of results.
This site can display upto 190 results.
Pharmacogenetics
|
February 18, 1999
An additional defective allele, CYP2C19*5, contributes to the S-mephenytoin poor metabolizer phenotype in Caucasians
G C Ibeanu, J Blaisdell, B I Ghanayem, et al.
Urologic Oncology
|
January 14, 2011
Environmental procarcinogen hypothesis of bladder cancer in humans: Dapsone hydroxylation as a susceptibility risk factor for aggressive bladder cancer
R Persad, C Fleming, H D Chern, et al.
Clinical Pharmacology and Therapeutics
|
September 1, 1990
Scleroderma is associated with differences in individual routes of drug metabolism: a study with dapsone, debrisoquin, and mephenytoin
D G May, C M Black, N J Olsen, et al.
Pharmacogenetics
|
January 1, 1995
The procarcinogen hypothesis for bladder cancer: activities of individual drug metabolizing enzymes as risk factors
R A Branch, H D Chern, A Adedoyin, et al.
Clinical Pharmacology and Therapeutics
|
August 1, 1986
Encainide disposition in patients with chronic cirrhosis
R H Bergstrand, T Wang, D M Roden, et al.
The Journal of Pharmacology and Experimental Therapeutics
|
January 22, 1998
A new genetic defect in human CYP2C19: mutation of the initiation codon is responsible for poor metabolism of S-mephenytoin
R J Ferguson, S M De Morais, S Benhamou, et al.
Pharmaceutical Research
|
April 23, 1999
Interrelationship between substrates and inhibitors of human CYP3A and P-glycoprotein
R B Kim, C Wandel, B Leake, et al.
Pharmacogenetics
|
December 12, 2001
In-vivo phenotyping for CYP3A by a single-point determination of midazolam plasma concentration
Y S Lin, G F Lockwood, M A Graham, et al.
Clinical Pharmacology and Therapeutics
|
August 15, 2001
Identification of functionally variant MDR1 alleles among European Americans and African Americans
R B Kim, B F Leake, E F Choo, et al.
AIDS Research and Human Retroviruses
|
October 29, 2000
Evidence of a source of HIV type 1 within the central nervous system by ultraintensive sampling of cerebrospinal fluid and plasma
D W Haas, L A Clough, B W Johnson, et al.
Page
of 19