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Journal of Cancer Research and Clinical Oncology
|
April 12, 1979
Early mitochondrial damage in the induction of haemorrhagic necrosis in the Crocker sarcoma (S 180) by endotoxin
G R Jones
Physiological Chemistry and Physics and Medical NMR
|
January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part I. The endogenous mechanism of self-destruction within the malignant cell, and the roles of endotoxin, certain hormones and drugs, and active oxygen in causing cellular injury and death
G R Jones
Journal of General Microbiology
|
July 1, 1975
A comparison of analytical methods for the numerical taxonomy of yeasts
G R Jones
Medical Hypotheses
|
January 3, 2001
Causes of Alzheimer's disease: paracetamol (acetaminophen) today? Amphetamines tomorrow?
G R Jones
Physiological Chemistry and Physics and Medical NMR
|
January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part III. Spontaneous drug resistance, selectivity of antineoplastic action, and strategies for intensifying tumor injury
G R Jones
Free Radical Research Communications
|
January 1, 1987
Dysbaric osteonecrosis (caisson disease of bone): are active oxygen species and the endocrine system responsible, and can control of the production of free radicals and their reaction products confer protection?
G R Jones
Medical Hypotheses
|
March 1, 1976
Cancer: restriction of chemical energy by fatty acids as the common pathway whereby various anti-tumour procedures selectively damage malignant cells in situ
G R Jones
Journal of the Canadian Association of Radiologists
|
March 1, 1983
Renal calculus ileus
G R Jones
Oncology
|
January 1, 1983
Interference with energy production as a means of causing injury to a human pancreatic carcinoma
G R Jones
Physiological Chemistry and Physics and Medical NMR
|
January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part II. Lipid peroxidation and cell death; drug resistance as a consequence of reversible cellular injury
G R Jones
Page
of 16
Search research articles
Search
Showing results (1-10 of 155) with videos related to
Sort By:
Page
of 16
Journal of Cancer Research and Clinical Oncology
|
April 12, 1979
Early mitochondrial damage in the induction of haemorrhagic necrosis in the Crocker sarcoma (S 180) by endotoxin
G R Jones
Physiological Chemistry and Physics and Medical NMR
|
January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part I. The endogenous mechanism of self-destruction within the malignant cell, and the roles of endotoxin, certain hormones and drugs, and active oxygen in causing cellular injury and death
G R Jones
Journal of General Microbiology
|
July 1, 1975
A comparison of analytical methods for the numerical taxonomy of yeasts
G R Jones
Medical Hypotheses
|
January 3, 2001
Causes of Alzheimer's disease: paracetamol (acetaminophen) today? Amphetamines tomorrow?
G R Jones
Physiological Chemistry and Physics and Medical NMR
|
January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part III. Spontaneous drug resistance, selectivity of antineoplastic action, and strategies for intensifying tumor injury
G R Jones
Free Radical Research Communications
|
January 1, 1987
Dysbaric osteonecrosis (caisson disease of bone): are active oxygen species and the endocrine system responsible, and can control of the production of free radicals and their reaction products confer protection?
G R Jones
Medical Hypotheses
|
March 1, 1976
Cancer: restriction of chemical energy by fatty acids as the common pathway whereby various anti-tumour procedures selectively damage malignant cells in situ
G R Jones
Journal of the Canadian Association of Radiologists
|
March 1, 1983
Renal calculus ileus
G R Jones
Oncology
|
January 1, 1983
Interference with energy production as a means of causing injury to a human pancreatic carcinoma
G R Jones
Physiological Chemistry and Physics and Medical NMR
|
January 1, 1992
Cancer destruction in vivo through disrupted energy metabolism. Part II. Lipid peroxidation and cell death; drug resistance as a consequence of reversible cellular injury
G R Jones
Page
of 16