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G Sankaran

Showing results (111-120 of 240) with videos related to

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Science (New York, N.Y.)|October 15, 2011
Correction of sickle cell disease in adult mice by interference with fetal hemoglobin silencingJian Xu, Cong Peng, Vijay G Sankaran, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 1, 2008
DNA polymorphisms at the BCL11A, HBS1L-MYB, and beta-globin loci associate with fetal hemoglobin levels and pain crises in sickle cell diseaseGuillaume Lettre, Vijay G Sankaran, Marcos André C Bezerra, et al.
Science (New York, N.Y.)|December 6, 2008
Human fetal hemoglobin expression is regulated by the developmental stage-specific repressor BCL11AVijay G Sankaran, Tobias F Menne, Jian Xu, et al.
British Journal of Haematology|March 17, 2022
A novel missense mutation outside the DNAJ domain of DNAJC21 is associated with Shwachman-Diamond syndromeMohammad Bilal Alsavaf, Jeffrey M Verboon, Muhammet E Dogan, et al.
Blood|December 16, 2010
Therapeutic levels of fetal hemoglobin in erythroid progeny of β-thalassemic CD34+ cells after lentiviral vector-mediated gene transferAndrew Wilber, Phillip W Hargrove, Yoon-Sang Kim, et al.
The Journal of Clinical Investigation|June 19, 2012
Exome sequencing identifies GATA1 mutations resulting in Diamond-Blackfan anemiaVijay G Sankaran, Roxanne Ghazvinian, Ron Do, et al.
The Lancet. Haematology|January 2, 2026
Management of transfusion-dependent β-thalassaemia in the era of novel therapies: a prioritisation-based matrix for settings with limited resourcesKhaled M Musallam, Sujit Sheth, Maria Domenica Cappellini, et al.
Biorxiv : the Preprint Server for Biology|September 24, 2024
Transcription factor networks disproportionately enrich for heritability of blood cell phenotypesJorge Diego Martin-Rufino, Alexis Caulier, Seayoung Lee, et al.
Science (New York, N.Y.)|April 3, 2025
Transcription factor networks disproportionately enrich for heritability of blood cell phenotypesJorge Diego Martin-Rufino, Alexis Caulier, Seayoung Lee, et al.
HGG Advances|January 20, 2022
From GWAS variant to function: A study of ∼148,000 variants for blood cell traitsQuan Sun, Cheynna A Crowley, Le Huang, et al.
Pageof 24

Showing results (111-120 of 240) with videos related to

Sort By:
Pageof 24
Science (New York, N.Y.)|October 15, 2011
Correction of sickle cell disease in adult mice by interference with fetal hemoglobin silencingJian Xu, Cong Peng, Vijay G Sankaran, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 1, 2008
DNA polymorphisms at the BCL11A, HBS1L-MYB, and beta-globin loci associate with fetal hemoglobin levels and pain crises in sickle cell diseaseGuillaume Lettre, Vijay G Sankaran, Marcos André C Bezerra, et al.
Science (New York, N.Y.)|December 6, 2008
Human fetal hemoglobin expression is regulated by the developmental stage-specific repressor BCL11AVijay G Sankaran, Tobias F Menne, Jian Xu, et al.
British Journal of Haematology|March 17, 2022
A novel missense mutation outside the DNAJ domain of DNAJC21 is associated with Shwachman-Diamond syndromeMohammad Bilal Alsavaf, Jeffrey M Verboon, Muhammet E Dogan, et al.
Blood|December 16, 2010
Therapeutic levels of fetal hemoglobin in erythroid progeny of β-thalassemic CD34+ cells after lentiviral vector-mediated gene transferAndrew Wilber, Phillip W Hargrove, Yoon-Sang Kim, et al.
The Journal of Clinical Investigation|June 19, 2012
Exome sequencing identifies GATA1 mutations resulting in Diamond-Blackfan anemiaVijay G Sankaran, Roxanne Ghazvinian, Ron Do, et al.
The Lancet. Haematology|January 2, 2026
Management of transfusion-dependent β-thalassaemia in the era of novel therapies: a prioritisation-based matrix for settings with limited resourcesKhaled M Musallam, Sujit Sheth, Maria Domenica Cappellini, et al.
Biorxiv : the Preprint Server for Biology|September 24, 2024
Transcription factor networks disproportionately enrich for heritability of blood cell phenotypesJorge Diego Martin-Rufino, Alexis Caulier, Seayoung Lee, et al.
Science (New York, N.Y.)|April 3, 2025
Transcription factor networks disproportionately enrich for heritability of blood cell phenotypesJorge Diego Martin-Rufino, Alexis Caulier, Seayoung Lee, et al.
HGG Advances|January 20, 2022
From GWAS variant to function: A study of ∼148,000 variants for blood cell traitsQuan Sun, Cheynna A Crowley, Le Huang, et al.
Pageof 24