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G Stucke

Showing results (41-50 of 58) with videos related to

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Anesthesiology|November 26, 2003
Halothane enhances gamma-aminobutyric acid receptor type A function but does not change overall inhibition in inspiratory premotor neurons in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Viseslav Tonkovic-Capin, et al.
Journal of Neurophysiology|November 13, 2009
Clinically relevant infusion rates of mu-opioid agonist remifentanil cause bradypnea in decerebrate dogs but not via direct effects in the pre-Bötzinger complex regionSanda Mustapic, Tomislav Radocaj, Antonio Sanchez, et al.
Journal of Neurophysiology|August 10, 2012
Pontine μ-opioid receptors mediate bradypnea caused by intravenous remifentanil infusions at clinically relevant concentrations in dogsIvana Prkic, Sanda Mustapic, Tomislav Radocaj, et al.
Anesthesiology|August 17, 2001
Effects of sevoflurane on excitatory neurotransmission to medullary expiratory neurons and on phrenic nerve activity in a decerebrate dog modelA G Stucke, E A Stuth, V Tonkovic-Capin, et al.
Journal of Neurophysiology|February 8, 2003
Differential processing of excitation by GABAergic gain modulation in canine caudal ventral respiratory group neuronsV Tonkovic-Capin, A G Stucke, E A Stuth, et al.
Anesthesiology|June 29, 2005
Sevoflurane depresses glutamatergic neurotransmission to brainstem inspiratory premotor neurons but not postsynaptic receptor function in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Viseslav Tonkovic-Capin, et al.
Anesthesiology|June 29, 2005
Sevoflurane enhances gamma-aminobutyric acid type A receptor function and overall inhibition of inspiratory premotor neurons in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Mirko Krolo, et al.
Anesthesiology|April 20, 2002
Effects of halothane and sevoflurane on inhibitory neurotransmission to medullary expiratory neurons in a decerebrate dog modelAstrid G Stucke, Eckehard A E Stuth, Viseslav Tonkovic-Capin, et al.
Anesthesiology|March 27, 2003
Halothane depresses glutamatergic neurotransmission to brain stem inspiratory premotor neurons in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Viseslav Tonkovic-Capin, et al.
Respiratory Physiology & Neurobiology|February 6, 2022
Contribution of the caudal medullary raphe to opioid induced respiratory depressionBarbara Palkovic, Denise Cook-Snyder, Jennifer J Callison, et al.
Pageof 6

Showing results (41-50 of 58) with videos related to

Sort By:
Pageof 6
Anesthesiology|November 26, 2003
Halothane enhances gamma-aminobutyric acid receptor type A function but does not change overall inhibition in inspiratory premotor neurons in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Viseslav Tonkovic-Capin, et al.
Journal of Neurophysiology|November 13, 2009
Clinically relevant infusion rates of mu-opioid agonist remifentanil cause bradypnea in decerebrate dogs but not via direct effects in the pre-Bötzinger complex regionSanda Mustapic, Tomislav Radocaj, Antonio Sanchez, et al.
Journal of Neurophysiology|August 10, 2012
Pontine μ-opioid receptors mediate bradypnea caused by intravenous remifentanil infusions at clinically relevant concentrations in dogsIvana Prkic, Sanda Mustapic, Tomislav Radocaj, et al.
Anesthesiology|August 17, 2001
Effects of sevoflurane on excitatory neurotransmission to medullary expiratory neurons and on phrenic nerve activity in a decerebrate dog modelA G Stucke, E A Stuth, V Tonkovic-Capin, et al.
Journal of Neurophysiology|February 8, 2003
Differential processing of excitation by GABAergic gain modulation in canine caudal ventral respiratory group neuronsV Tonkovic-Capin, A G Stucke, E A Stuth, et al.
Anesthesiology|June 29, 2005
Sevoflurane depresses glutamatergic neurotransmission to brainstem inspiratory premotor neurons but not postsynaptic receptor function in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Viseslav Tonkovic-Capin, et al.
Anesthesiology|June 29, 2005
Sevoflurane enhances gamma-aminobutyric acid type A receptor function and overall inhibition of inspiratory premotor neurons in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Mirko Krolo, et al.
Anesthesiology|April 20, 2002
Effects of halothane and sevoflurane on inhibitory neurotransmission to medullary expiratory neurons in a decerebrate dog modelAstrid G Stucke, Eckehard A E Stuth, Viseslav Tonkovic-Capin, et al.
Anesthesiology|March 27, 2003
Halothane depresses glutamatergic neurotransmission to brain stem inspiratory premotor neurons in a decerebrate dog modelAstrid G Stucke, Edward J Zuperku, Viseslav Tonkovic-Capin, et al.
Respiratory Physiology & Neurobiology|February 6, 2022
Contribution of the caudal medullary raphe to opioid induced respiratory depressionBarbara Palkovic, Denise Cook-Snyder, Jennifer J Callison, et al.
Pageof 6