G W Dorn|M G Davis - JoVE Visualize

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Circulation|May 12, 1998

Effects of total replacement of atrial myosin light chain-2 with the ventricular isoform in atrial myocytes of transgenic miceC M Pawloski-Dahm, G Song, D L Kirkpatrick, et al.

Molecular Pharmacology|July 11, 1998

Phosphorylation and functional desensitization of the alpha2A-adrenergic receptor by protein kinase CM Liang, M G Eason, E A Jewell-Motz, et al.

The Journal of Biological Chemistry|July 20, 1999

Cardiac specific overexpression of transglutaminase II (G(h)) results in a unique hypertrophy phenotype independent of phospholipase C activationK Small, J F Feng, J Lorenz, et al.

Circulation Research|June 13, 2000

Cardiotrophic effects of protein kinase C epsilon: analysis by in vivo modulation of PKCepsilon translocationD Mochly-Rosen, G Wu, H Hahn, et al.

The Journal of Clinical Investigation|June 22, 1999

Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failureV P Sah, S Minamisawa, S P Tam, et al.

Circulation Research|February 19, 2000

Calcineurin-mediated hypertrophy protects cardiomyocytes from apoptosis in vitro and in vivo: An apoptosis-independent model of dilated heart failureL J De Windt, H W Lim, T Taigen, et al.

Circulation|April 12, 2000

Early and delayed consequences of beta(2)-adrenergic receptor overexpression in mouse hearts: critical role for expression levelS B Liggett, N M Tepe, J N Lorenz, et al.

International Journal of Food Microbiology|November 10, 2010

A dose dependent impact of prebiotic galactooligosaccharides on the intestinal microbiota of healthy adultsL M G Davis, I Martínez, J Walter, et al.

Circulation|May 23, 2001

Mouse model of desmin-related cardiomyopathyX Wang, H Osinska, G W Dorn, et al.

The Journal of Biological Chemistry|October 17, 1998

Cardiac-specific overexpression of mouse cardiac calsequestrin is associated with depressed cardiovascular function and hypertrophy in transgenic miceY Sato, D G Ferguson, H Sako, et al.

Pageof 12

Showing results (71-80 of 119) with videos related to

Sort By:

Pageof 12

Circulation|May 12, 1998

Effects of total replacement of atrial myosin light chain-2 with the ventricular isoform in atrial myocytes of transgenic miceC M Pawloski-Dahm, G Song, D L Kirkpatrick, et al.

Molecular Pharmacology|July 11, 1998

Phosphorylation and functional desensitization of the alpha2A-adrenergic receptor by protein kinase CM Liang, M G Eason, E A Jewell-Motz, et al.

The Journal of Biological Chemistry|July 20, 1999

Cardiac specific overexpression of transglutaminase II (G(h)) results in a unique hypertrophy phenotype independent of phospholipase C activationK Small, J F Feng, J Lorenz, et al.

Circulation Research|June 13, 2000

Cardiotrophic effects of protein kinase C epsilon: analysis by in vivo modulation of PKCepsilon translocationD Mochly-Rosen, G Wu, H Hahn, et al.

The Journal of Clinical Investigation|June 22, 1999

Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failureV P Sah, S Minamisawa, S P Tam, et al.

Circulation Research|February 19, 2000

Calcineurin-mediated hypertrophy protects cardiomyocytes from apoptosis in vitro and in vivo: An apoptosis-independent model of dilated heart failureL J De Windt, H W Lim, T Taigen, et al.

Circulation|April 12, 2000

Early and delayed consequences of beta(2)-adrenergic receptor overexpression in mouse hearts: critical role for expression levelS B Liggett, N M Tepe, J N Lorenz, et al.

International Journal of Food Microbiology|November 10, 2010

A dose dependent impact of prebiotic galactooligosaccharides on the intestinal microbiota of healthy adultsL M G Davis, I Martínez, J Walter, et al.

Circulation|May 23, 2001

Mouse model of desmin-related cardiomyopathyX Wang, H Osinska, G W Dorn, et al.

The Journal of Biological Chemistry|October 17, 1998

Cardiac-specific overexpression of mouse cardiac calsequestrin is associated with depressed cardiovascular function and hypertrophy in transgenic miceY Sato, D G Ferguson, H Sako, et al.

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