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Research Communications in Chemical Pathology and Pharmacology|August 1, 1983
Release of acetylcholine from fragments of human term placenta and effects of the choline acetyltransferase inhibitor (2-benzoylethyl)trimethylammonium iodideF Welsch, D B Stedman, W C WengerExperimental Cell Research|July 1, 1985
Effects of a teratogen on [3H]uridine nucleotide transfer between human embryonal cells and on gap junctionsF Welsch, D B Stedman, J L CarsonPlacenta. Supplement|January 1, 1981
Acetylcholine in human term placenta: tissue levels in intact fragments after inhibition in vitro of choline acetyltransferase and relationship to [14C]alpha-aminoisobutyric acid uptakeF Welsch, W C Wenger, D B StedmanReproductive Toxicology (Elmsford, N.Y.)|July 26, 2000
Effect of different sampling designs on outcome of endocrine disruptor studiesB A Elswick, F Welsch, D B JanszenToxicology and Applied Pharmacology|April 1, 1988
The relationship of embryotoxicity to disposition of 2-methoxyethanol in miceR B Sleet, J A Greene, F WelschJournal of Biochemical Toxicology|January 1, 1987
Attenuation of 2-methoxyethanol and methoxyacetic acid-induced digit malformations in mice by simple physiological compounds: implications for the role of further metabolism of methoxyacetic acid in developmental toxicityF Welsch, R B Sleet, J A GreeneToxicology Letters|December 1, 1995
Physiologically based pharmacokinetic models applicable to organogenesis: extrapolation between species and potential use in prenatal toxicity risk assessmentsF Welsch, G M Blumenthal, R B ConollyToxicology and Applied Pharmacology|May 1, 1992
2-Methoxyacetic acid dosimetry-teratogenicity relationships in CD-1 mice exposed to 2-methoxyethanolD O Clarke, J M Duignan, F WelschToxicology and Applied Pharmacology|June 1, 1989
Attenuation of 2-methoxyethanol-induced testicular toxicity in the rat by simple physiological compoundsC A Mebus, F Welsch, P K WorkingPlacenta|July 1, 1981
Choline metabolism in placenta: evidence for the biosynthesis of phosphatidylcholine in microsomes via the methylation pathwayF Welsch, W C Wenger, D B StedmanPageof 196