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Gabriel Stölting

Showing results (21-30 of 30) with videos related to

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Pflugers Archiv : European Journal of Physiology|May 2, 2013
Regulation of ClC-2 gating by intracellular ATPGabriel Stölting, Georgeta Teodorescu, Birgit Begemann, et al.
The Journal of Biological Chemistry|December 24, 2014
Direct interaction of CaVβ with actin up-regulates L-type calcium currents in HL-1 cardiomyocytesGabriel Stölting, Regina Campos de Oliveira, Raul E Guzman, et al.
Elife|December 10, 2015
A K(+)-selective CNG channel orchestrates Ca(2+) signalling in zebrafish spermSylvia Fechner, Luis Alvarez, Wolfgang Bönigk, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 21, 2021
Enhanced Ca<sup>2+</sup> signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (<i>Cacna1h</i><sup></sup> )Eric Seidel, Julia Schewe, Junhui Zhang, et al.
JCI Insight|September 12, 2023
Isradipine therapy in Cacna1dIle772Met/+ mice ameliorates primary aldosteronism and neurologic abnormalitiesGabriel Stölting, Hoang An Dinh, Marina Volkert, et al.
Neurology. Genetics|September 9, 2024
A Novel De Novo Gain-of-Function <i>CACNA1D</i> Variant in Neurodevelopmental Disease With Congenital Tremor, Seizures, and HypotoniaFabian Dannenberg, Arpad Von Moers, Petra Bittigau, et al.
British Journal of Pharmacology|May 4, 2018
A novel cross-species inhibitor to study the function of CatSper Ca<sup>2+</sup> channels in spermAndreas Rennhack, Christian Schiffer, Christoph Brenker, et al.
Elife|April 25, 2015
Recurrent gain of function mutation in calcium channel CACNA1H causes early-onset hypertension with primary aldosteronismUte I Scholl, Gabriel Stölting, Carol Nelson-Williams, et al.
Nature Genetics|February 7, 2018
CLCN2 chloride channel mutations in familial hyperaldosteronism type IIUte I Scholl, Gabriel Stölting, Julia Schewe, et al.
Nature Genetics|August 6, 2013
Somatic and germline CACNA1D calcium channel mutations in aldosterone-producing adenomas and primary aldosteronismUte I Scholl, Gerald Goh, Gabriel Stölting, et al.
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Showing results (21-30 of 30) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 30 results.
Pflugers Archiv : European Journal of Physiology|May 2, 2013
Regulation of ClC-2 gating by intracellular ATPGabriel Stölting, Georgeta Teodorescu, Birgit Begemann, et al.
The Journal of Biological Chemistry|December 24, 2014
Direct interaction of CaVβ with actin up-regulates L-type calcium currents in HL-1 cardiomyocytesGabriel Stölting, Regina Campos de Oliveira, Raul E Guzman, et al.
Elife|December 10, 2015
A K(+)-selective CNG channel orchestrates Ca(2+) signalling in zebrafish spermSylvia Fechner, Luis Alvarez, Wolfgang Bönigk, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 21, 2021
Enhanced Ca<sup>2+</sup> signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (<i>Cacna1h</i><sup></sup> )Eric Seidel, Julia Schewe, Junhui Zhang, et al.
JCI Insight|September 12, 2023
Isradipine therapy in Cacna1dIle772Met/+ mice ameliorates primary aldosteronism and neurologic abnormalitiesGabriel Stölting, Hoang An Dinh, Marina Volkert, et al.
Neurology. Genetics|September 9, 2024
A Novel De Novo Gain-of-Function <i>CACNA1D</i> Variant in Neurodevelopmental Disease With Congenital Tremor, Seizures, and HypotoniaFabian Dannenberg, Arpad Von Moers, Petra Bittigau, et al.
British Journal of Pharmacology|May 4, 2018
A novel cross-species inhibitor to study the function of CatSper Ca<sup>2+</sup> channels in spermAndreas Rennhack, Christian Schiffer, Christoph Brenker, et al.
Elife|April 25, 2015
Recurrent gain of function mutation in calcium channel CACNA1H causes early-onset hypertension with primary aldosteronismUte I Scholl, Gabriel Stölting, Carol Nelson-Williams, et al.
Nature Genetics|February 7, 2018
CLCN2 chloride channel mutations in familial hyperaldosteronism type IIUte I Scholl, Gabriel Stölting, Julia Schewe, et al.
Nature Genetics|August 6, 2013
Somatic and germline CACNA1D calcium channel mutations in aldosterone-producing adenomas and primary aldosteronismUte I Scholl, Gerald Goh, Gabriel Stölting, et al.
Pageof 3