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Neuro-Degenerative Diseases|February 18, 2010
Are Abeta and its derivatives causative agents or innocent bystanders in AD?Nikolaos K Robakis
Amyloid : the International Journal of Experimental and Clinical Investigation : the Official Journal of the International Society of Amyloidosis|September 11, 2003
An Alzheimer's disease hypothesis based on transcriptional dysregulationNikolaos K Robakis
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|September 1, 2017
Presenilin1/γ-secretase protects neurons from glucose deprivation-induced death by regulating miR-212 and PEA15Qian Huang, Georgios Voloudakis, Yimin Ren, et al.
Progress in Neurobiology|May 20, 2020
Presynaptic failure in Alzheimer's diseaseGael Barthet, Christophe Mulle
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 20, 2015
Presenilin 1 is necessary for neuronal, but not glial, EGFR expression and neuroprotection via γ-secretase-independent transcriptional mechanismsJulien Bruban, Georgios Voloudakis, Qian Huang, et al.
Molecular Psychiatry|June 17, 2020
PS1 FAD mutants decrease ephrinB2-regulated angiogenic functions, ischemia-induced brain neovascularization and neuronal survivalYoneJung Yoon, Georgios Voloudakis, Nathan Doran, et al.
Neuro-Degenerative Diseases|June 29, 2007
Novel processing of Notch 1 within its intracellular domain by a cysteine proteaseAngeliki Fassa, Loukia Parisiadou, Nikolaos K Robakis, et al.
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