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[Rinsho Ketsueki] the Japanese Journal of Clinical Hematology|October 2, 2024
[Toward optimizing AML treatment in the elderly]Gaku OshikawaCancers|May 16, 2023
Optimizing Treatment Options for Newly Diagnosed Acute Myeloid Leukemia in Older Patients with ComorbiditiesGaku Oshikawa, Koji SasakiPlos One|November 22, 2013
Inhibition of the PI3K/Akt/GSK3 pathway downstream of BCR/ABL, Jak2-V617F, or FLT3-ITD downregulates DNA damage-induced Chk1 activation as well as G2/M arrest and prominently enhances induction of apoptosisTetsuya Kurosu, Toshikage Nagao, Nan Wu, et al.International Journal of Oncology|December 13, 2012
PECAM-1 is involved in BCR/ABL signaling and may downregulate imatinib-induced apoptosis of Philadelphia chromosome-positive leukemia cellsNan Wu, Tetsuya Kurosu, Gaku Oshikawa, et al.Plos One|November 17, 2011
DNA damage stress and inhibition of Jak2-V617F cause its degradation and synergistically induce apoptosis through activation of GSK3βToshikage Nagao, Gaku Oshikawa, Nan Wu, et al.[Rinsho Ketsueki] the Japanese Journal of Clinical Hematology|June 3, 2026
[Autoimmune acquired factor V deficiency developing after thoracic aortic aneurysm repair]Yuya Sasaki, Hajime Murakami, Takuma Harada, et al.[Rinsho Ketsueki] the Japanese Journal of Clinical Hematology|March 7, 2009
[Primary multifocal osseous Hodgkin lymphoma]Gaku Oshikawa, Ayako Arai, Koji Sasaki, et al.[Rinsho Ketsueki] the Japanese Journal of Clinical Hematology|July 8, 2026
[Immune-mediated thrombotic thrombocytopenic purpura with recurrent remission achieved by fresh frozen plasma transfusion and steroid therapy without plasma exchange]Asuka Kono, Miwa Watanabe, Yuya Sasaki, et al.Apoptosis : an International Journal on Programmed Cell Death|January 23, 2010
Enhancement of imatinib-induced apoptosis of BCR/ABL-expressing cells by nutlin-3 through synergistic activation of the mitochondrial apoptotic pathwayTetsuya Kurosu, Nan Wu, Gaku Oshikawa, et al.Cancer Genetics and Cytogenetics|April 27, 2010
Clonal evolution with double Ph followed by tetraploidy in imatinib-treated chronic myeloid leukemia with e19a2 transcript in transformationGaku Oshikawa, Tetsuya Kurosu, Ayako Arai, et al.Pageof 4