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Gareth Banks

Showing results (11-20 of 24) with videos related to

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The Journal of Biological Chemistry|October 5, 2010
Neurodegenerative mutation in cytoplasmic dynein alters its organization and dynein-dynactin and dynein-kinesin interactionsWenhan Deng, Caroline Garrett, Benjamin Dombert, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|September 15, 2023
A missense mutation in zinc finger homeobox-3 (ZFHX3) impedes growth and alters metabolism and hypothalamic gene expression in micePatrick M Nolan, Gareth Banks, Nora Bourbia, et al.
Neurobiology of Aging|September 3, 2014
Genetic background influences age-related decline in visual and nonvisual retinal responses, circadian rhythms, and sleepGareth Banks, Ines Heise, Becky Starbuck, et al.
Iscience|October 11, 2021
Zfhx3-mediated genetic ablation of the SCN abolishes light entrainable circadian activity while sparing food anticipatory activityAshleigh G Wilcox, R Sonia Bains, Debbie Williams, et al.
Disease Models & Mechanisms|August 26, 2025
Multi-modal comparative phenotyping of knock-in mouse models of frontotemporal dementia/amyotrophic lateral sclerosisSevda Boyanova, Gareth Banks, Tatiana V Lipina, et al.
Mechanisms of Ageing and Development|October 6, 2018
Modelling physical resilience in ageing miceMarkus Schosserer, Gareth Banks, Soner Dogan, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 12, 2021
Zfhx3 modulates retinal sensitivity and circadian responses to lightSteven Hughes, Jessica K Edwards, Ashleigh G Wilcox, et al.
The Journal of Biological Chemistry|April 13, 2010
The legs at odd angles (Loa) mutation in cytoplasmic dynein ameliorates mitochondrial function in SOD1G93A mouse model for motor neuron diseaseAli Morsi El-Kadi, Virginie Bros-Facer, Wenhan Deng, et al.
Disease Models & Mechanisms|January 30, 2019
Loss of <i>Frrs1l</i> disrupts synaptic AMPA receptor function, and results in neurodevelopmental, motor, cognitive and electrographical abnormalitiesMichelle Stewart, Petrina Lau, Gareth Banks, et al.
International Journal of Molecular Sciences|January 21, 2022
Tissue Proteome of 2-Hydroxyacyl-CoA Lyase Deficient Mice Reveals Peroxisome Proliferation and Activation of ω-OxidationYoussef Khalil, Sara Carrino, Fujun Lin, et al.
Pageof 3

Showing results (11-20 of 24) with videos related to

Sort By:
Pageof 3
The Journal of Biological Chemistry|October 5, 2010
Neurodegenerative mutation in cytoplasmic dynein alters its organization and dynein-dynactin and dynein-kinesin interactionsWenhan Deng, Caroline Garrett, Benjamin Dombert, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|September 15, 2023
A missense mutation in zinc finger homeobox-3 (ZFHX3) impedes growth and alters metabolism and hypothalamic gene expression in micePatrick M Nolan, Gareth Banks, Nora Bourbia, et al.
Neurobiology of Aging|September 3, 2014
Genetic background influences age-related decline in visual and nonvisual retinal responses, circadian rhythms, and sleepGareth Banks, Ines Heise, Becky Starbuck, et al.
Iscience|October 11, 2021
Zfhx3-mediated genetic ablation of the SCN abolishes light entrainable circadian activity while sparing food anticipatory activityAshleigh G Wilcox, R Sonia Bains, Debbie Williams, et al.
Disease Models & Mechanisms|August 26, 2025
Multi-modal comparative phenotyping of knock-in mouse models of frontotemporal dementia/amyotrophic lateral sclerosisSevda Boyanova, Gareth Banks, Tatiana V Lipina, et al.
Mechanisms of Ageing and Development|October 6, 2018
Modelling physical resilience in ageing miceMarkus Schosserer, Gareth Banks, Soner Dogan, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 12, 2021
Zfhx3 modulates retinal sensitivity and circadian responses to lightSteven Hughes, Jessica K Edwards, Ashleigh G Wilcox, et al.
The Journal of Biological Chemistry|April 13, 2010
The legs at odd angles (Loa) mutation in cytoplasmic dynein ameliorates mitochondrial function in SOD1G93A mouse model for motor neuron diseaseAli Morsi El-Kadi, Virginie Bros-Facer, Wenhan Deng, et al.
Disease Models & Mechanisms|January 30, 2019
Loss of <i>Frrs1l</i> disrupts synaptic AMPA receptor function, and results in neurodevelopmental, motor, cognitive and electrographical abnormalitiesMichelle Stewart, Petrina Lau, Gareth Banks, et al.
International Journal of Molecular Sciences|January 21, 2022
Tissue Proteome of 2-Hydroxyacyl-CoA Lyase Deficient Mice Reveals Peroxisome Proliferation and Activation of ω-OxidationYoussef Khalil, Sara Carrino, Fujun Lin, et al.
Pageof 3