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Gareth R Howell

Showing results (101-110 of 127) with videos related to

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Alzheimer'S & Dementia (New York, N. Y.)|January 18, 2021
Translational animal models for Alzheimer's disease: An Alzheimer's Association Business Consortium Think TankMichael P Vitek, Joseph A Araujo, Michael Fossel, et al.
The Journal of Clinical Investigation|March 20, 2012
Radiation treatment inhibits monocyte entry into the optic nerve head and prevents neuronal damage in a mouse model of glaucomaGareth R Howell, Ileana Soto, Xianjun Zhu, et al.
Cell|December 18, 2007
The classical complement cascade mediates CNS synapse eliminationBeth Stevens, Nicola J Allen, Luis E Vazquez, et al.
Plos Genetics|May 10, 2014
A dominant-negative mutation of mouse Lmx1b causes glaucoma and is semi-lethal via LDB1-mediated dimerization [corrected]Sally H Cross, Danilo G Macalinao, Lisa McKie, et al.
Alzheimer'S & Dementia (New York, N. Y.)|December 7, 2020
Model organism development and evaluation for late-onset Alzheimer's disease: MODEL-ADAdrian L Oblak, Stefania Forner, Paul R Territo, et al.
Developmental Biology|April 1, 2006
A robust system for RNA interference in the chicken using a modified microRNA operonRaman M Das, Nick J Van Hateren, Gareth R Howell, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|June 16, 2026
WSB.APP/PS1 mice develop age-dependent cerebral amyloid angiopathy, cerebrovascular dysfunction, and white matter deficitsOlivia J Marola, Asli Uyar, Kelly J Keezer, et al.
The Journal of Experimental Medicine|March 4, 2015
TREM2 deficiency eliminates TREM2+ inflammatory macrophages and ameliorates pathology in Alzheimer's disease mouse modelsTaylor R Jay, Crystal M Miller, Paul J Cheng, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
WSB.<i>APP/PS1</i> mice develop age-dependent cerebral amyloid angiopathy, cerebrovascular deficits, and white matter damage, which are modified by humanized <i>APOE</i> allelesKristen D Onos, Olivia J Marola, Asli Uyar, et al.
Alzheimer'S & Dementia (New York, N. Y.)|March 12, 2024
New directions for Alzheimer's disease research from the Jackson Laboratory Center for Alzheimer's and Dementia Research 2022 workshopMaria A Telpoukhovskaia, Thomas J Murdy, Olivia J Marola, et al.
Pageof 13

Showing results (101-110 of 127) with videos related to

Sort By:
Pageof 13
Alzheimer'S & Dementia (New York, N. Y.)|January 18, 2021
Translational animal models for Alzheimer's disease: An Alzheimer's Association Business Consortium Think TankMichael P Vitek, Joseph A Araujo, Michael Fossel, et al.
The Journal of Clinical Investigation|March 20, 2012
Radiation treatment inhibits monocyte entry into the optic nerve head and prevents neuronal damage in a mouse model of glaucomaGareth R Howell, Ileana Soto, Xianjun Zhu, et al.
Cell|December 18, 2007
The classical complement cascade mediates CNS synapse eliminationBeth Stevens, Nicola J Allen, Luis E Vazquez, et al.
Plos Genetics|May 10, 2014
A dominant-negative mutation of mouse Lmx1b causes glaucoma and is semi-lethal via LDB1-mediated dimerization [corrected]Sally H Cross, Danilo G Macalinao, Lisa McKie, et al.
Alzheimer'S & Dementia (New York, N. Y.)|December 7, 2020
Model organism development and evaluation for late-onset Alzheimer's disease: MODEL-ADAdrian L Oblak, Stefania Forner, Paul R Territo, et al.
Developmental Biology|April 1, 2006
A robust system for RNA interference in the chicken using a modified microRNA operonRaman M Das, Nick J Van Hateren, Gareth R Howell, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|June 16, 2026
WSB.APP/PS1 mice develop age-dependent cerebral amyloid angiopathy, cerebrovascular dysfunction, and white matter deficitsOlivia J Marola, Asli Uyar, Kelly J Keezer, et al.
The Journal of Experimental Medicine|March 4, 2015
TREM2 deficiency eliminates TREM2+ inflammatory macrophages and ameliorates pathology in Alzheimer's disease mouse modelsTaylor R Jay, Crystal M Miller, Paul J Cheng, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
WSB.<i>APP/PS1</i> mice develop age-dependent cerebral amyloid angiopathy, cerebrovascular deficits, and white matter damage, which are modified by humanized <i>APOE</i> allelesKristen D Onos, Olivia J Marola, Asli Uyar, et al.
Alzheimer'S & Dementia (New York, N. Y.)|March 12, 2024
New directions for Alzheimer's disease research from the Jackson Laboratory Center for Alzheimer's and Dementia Research 2022 workshopMaria A Telpoukhovskaia, Thomas J Murdy, Olivia J Marola, et al.
Pageof 13