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Gavin Rumbaugh

Showing results (51-60 of 81) with videos related to

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Nature Communications|January 18, 2025
Syngap1 promotes cognitive function through regulation of cortical sensorimotor dynamicsThomas Vaissiere, Sheldon D Michaelson, Thomas Creson, et al.
Nature Neuroscience|December 20, 2017
Publisher Correction: An interactive framework for whole-brain maps at cellular resolutionDaniel Fürth, Thomas Vaissière, Ourania Tzortzi, et al.
Nature Neuroscience|December 6, 2017
An interactive framework for whole-brain maps at cellular resolutionDaniel Fürth, Thomas Vaissière, Ourania Tzortzi, et al.
Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|April 4, 2015
Pharmacological Selectivity Within Class I Histone Deacetylases Predicts Effects on Synaptic Function and Memory RescueGavin Rumbaugh, Stephanie E Daws, Emin D Ozkan, et al.
Neuron|August 28, 2010
Myosin IIb regulates actin dynamics during synaptic plasticity and memory formationChristopher S Rex, Cristin F Gavin, Maria D Rubio, et al.
Cell|November 13, 2012
Pathogenic SYNGAP1 mutations impair cognitive development by disrupting maturation of dendritic spine synapsesJames P Clement, Massimiliano Aceti, Thomas K Creson, et al.
Biorxiv : the Preprint Server for Biology|June 9, 2023
Basolateral Amygdala Corticotrophin Releasing Factor Receptor 2 Interacts with Nonmuscle Myosin II to Destabilize MemoryMadalyn Hafenbreidel, Sherri B Briggs, Meghana Arza, et al.
Cell|March 12, 2003
Phosphorylation of the AMPA receptor GluR1 subunit is required for synaptic plasticity and retention of spatial memoryHey-Kyoung Lee, Kogo Takamiya, Jung-Soo Han, et al.
Nature Neuroscience|November 21, 2018
SYNGAP1 heterozygosity disrupts sensory processing by reducing touch-related activity within somatosensory cortex circuitsSheldon D Michaelson, Emin D Ozkan, Massimiliano Aceti, et al.
Neuron|June 20, 2014
Reduced cognition in Syngap1 mutants is caused by isolated damage within developing forebrain excitatory neuronsEmin D Ozkan, Thomas K Creson, Enikö A Kramár, et al.
Pageof 9

Showing results (51-60 of 81) with videos related to

Sort By:
Pageof 9
Nature Communications|January 18, 2025
Syngap1 promotes cognitive function through regulation of cortical sensorimotor dynamicsThomas Vaissiere, Sheldon D Michaelson, Thomas Creson, et al.
Nature Neuroscience|December 20, 2017
Publisher Correction: An interactive framework for whole-brain maps at cellular resolutionDaniel Fürth, Thomas Vaissière, Ourania Tzortzi, et al.
Nature Neuroscience|December 6, 2017
An interactive framework for whole-brain maps at cellular resolutionDaniel Fürth, Thomas Vaissière, Ourania Tzortzi, et al.
Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|April 4, 2015
Pharmacological Selectivity Within Class I Histone Deacetylases Predicts Effects on Synaptic Function and Memory RescueGavin Rumbaugh, Stephanie E Daws, Emin D Ozkan, et al.
Neuron|August 28, 2010
Myosin IIb regulates actin dynamics during synaptic plasticity and memory formationChristopher S Rex, Cristin F Gavin, Maria D Rubio, et al.
Cell|November 13, 2012
Pathogenic SYNGAP1 mutations impair cognitive development by disrupting maturation of dendritic spine synapsesJames P Clement, Massimiliano Aceti, Thomas K Creson, et al.
Biorxiv : the Preprint Server for Biology|June 9, 2023
Basolateral Amygdala Corticotrophin Releasing Factor Receptor 2 Interacts with Nonmuscle Myosin II to Destabilize MemoryMadalyn Hafenbreidel, Sherri B Briggs, Meghana Arza, et al.
Cell|March 12, 2003
Phosphorylation of the AMPA receptor GluR1 subunit is required for synaptic plasticity and retention of spatial memoryHey-Kyoung Lee, Kogo Takamiya, Jung-Soo Han, et al.
Nature Neuroscience|November 21, 2018
SYNGAP1 heterozygosity disrupts sensory processing by reducing touch-related activity within somatosensory cortex circuitsSheldon D Michaelson, Emin D Ozkan, Massimiliano Aceti, et al.
Neuron|June 20, 2014
Reduced cognition in Syngap1 mutants is caused by isolated damage within developing forebrain excitatory neuronsEmin D Ozkan, Thomas K Creson, Enikö A Kramár, et al.
Pageof 9