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Cardiovascular Research|January 15, 2015
Syndecan-4 is a key determinant of collagen cross-linking and passive myocardial stiffness in the pressure-overloaded heartKate M Herum, Ida G Lunde, Biljana Skrbic, et al.Scientific Reports|June 16, 2017
Wnt5a is associated with right ventricular dysfunction and adverse outcome in dilated cardiomyopathyAurelija Abraityte, Ida G Lunde, Erik T Askevold, et al.The FEBS Journal|March 14, 2013
Lumican is increased in experimental and clinical heart failure, and its production by cardiac fibroblasts is induced by mechanical and proinflammatory stimuliKristin V T Engebretsen, Ida G Lunde, Mari E Strand, et al.Mediators of Inflammation|May 12, 2017
Toll-Like Receptor 9 Promotes Survival in SERCA2a KO Heart Failure MiceYangchen Dhondup, Ivar Sjaastad, Øystein Sandanger, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|April 19, 2011
Lack of CCR7 induces pulmonary hypertension involving perivascular leukocyte infiltration and inflammationKarl-Otto Larsen, Arne Yndestad, Ivar Sjaastad, et al.Cardiovascular Research|February 20, 2015
Lack of collagen VIII reduces fibrosis and promotes early mortality and cardiac dilatation in pressure overload in miceBiljana Skrbic, Kristin V T Engebretsen, Mari E Strand, et al.American Journal of Physiology. Heart and Circulatory Physiology|April 28, 2023
Beneficial effects of exercise initiated before development of hypertrophic cardiomyopathy in genotype-positive miceKristine Andreassen, Chloe Rixon, Marie H Hansen, et al.European Heart Journal|March 31, 2009
Increased systemic and myocardial expression of neutrophil gelatinase-associated lipocalin in clinical and experimental heart failureArne Yndestad, Linn Landrø, Thor Ueland, et al.Scientific Reports|October 6, 2021
The extracellular matrix glycoprotein ADAMTSL2 is increased in heart failure and inhibits TGFβ signalling in cardiac fibroblastsKaroline B Rypdal, Pugazendhi M Erusappan, A Olav Melleby, et al.Circulation. Heart Failure|November 19, 2009
Increased production of CXCL16 in experimental and clinical heart failure: a possible role in extracellular matrix remodelingChristen Peder Dahl, Cathrine Husberg, Lars Gullestad, et al.Pageof 15