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George A Carlson

Showing results (41-50 of 72) with videos related to

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The American Journal of Pathology|August 23, 2011
Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brainKatherine J Kopeikina, George A Carlson, Rose Pitstick, et al.
Journal of Neurochemistry|May 19, 2004
Increased plaque burden in brains of APP mutant MnSOD heterozygous knockout miceFeng Li, Noel Y Calingasan, Fangmin Yu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 6, 2013
Innate immunity receptor CD36 promotes cerebral amyloid angiopathyLaibaik Park, Joan Zhou, Ping Zhou, et al.
The Journal of Comparative Neurology|July 11, 2013
Tau-amyloid interactions in the rTgTauEC model of early Alzheimer's disease suggest amyloid-induced disruption of axonal projections and exacerbated axonal pathologyAmy M Pooler, Manuela Polydoro, Susanne K Wegmann, et al.
Acta Neuropathologica|November 26, 2013
Soluble pathological tau in the entorhinal cortex leads to presynaptic deficits in an early Alzheimer's disease modelManuela Polydoro, Volodymyr I Dzhala, Amy M Pooler, et al.
Genetica|December 29, 2004
Implementing large-scale ENU mutagenesis screens in North AmericaAmander T Clark, Daniel Goldowitz, Joseph S Takahashi, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 9, 2011
Scavenger receptor CD36 is essential for the cerebrovascular oxidative stress and neurovascular dysfunction induced by amyloid-betaLaibaik Park, Gang Wang, Ping Zhou, et al.
Neuron|February 28, 2012
Propagation of tau pathology in a model of early Alzheimer's diseaseAlix de Calignon, Manuela Polydoro, Marc Suárez-Calvet, et al.
Neuron|December 22, 2010
Tau mislocalization to dendritic spines mediates synaptic dysfunction independently of neurodegenerationBrian R Hoover, Miranda N Reed, Jianjun Su, et al.
Disease Models & Mechanisms|February 8, 2017
The familial dysautonomia disease gene <i>IKBKAP</i> is required in the developing and adult mouse central nervous systemMarta Chaverra, Lynn George, Marc Mergy, et al.
Pageof 8

Showing results (41-50 of 72) with videos related to

Sort By:
Pageof 8
The American Journal of Pathology|August 23, 2011
Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brainKatherine J Kopeikina, George A Carlson, Rose Pitstick, et al.
Journal of Neurochemistry|May 19, 2004
Increased plaque burden in brains of APP mutant MnSOD heterozygous knockout miceFeng Li, Noel Y Calingasan, Fangmin Yu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 6, 2013
Innate immunity receptor CD36 promotes cerebral amyloid angiopathyLaibaik Park, Joan Zhou, Ping Zhou, et al.
The Journal of Comparative Neurology|July 11, 2013
Tau-amyloid interactions in the rTgTauEC model of early Alzheimer's disease suggest amyloid-induced disruption of axonal projections and exacerbated axonal pathologyAmy M Pooler, Manuela Polydoro, Susanne K Wegmann, et al.
Acta Neuropathologica|November 26, 2013
Soluble pathological tau in the entorhinal cortex leads to presynaptic deficits in an early Alzheimer's disease modelManuela Polydoro, Volodymyr I Dzhala, Amy M Pooler, et al.
Genetica|December 29, 2004
Implementing large-scale ENU mutagenesis screens in North AmericaAmander T Clark, Daniel Goldowitz, Joseph S Takahashi, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 9, 2011
Scavenger receptor CD36 is essential for the cerebrovascular oxidative stress and neurovascular dysfunction induced by amyloid-betaLaibaik Park, Gang Wang, Ping Zhou, et al.
Neuron|February 28, 2012
Propagation of tau pathology in a model of early Alzheimer's diseaseAlix de Calignon, Manuela Polydoro, Marc Suárez-Calvet, et al.
Neuron|December 22, 2010
Tau mislocalization to dendritic spines mediates synaptic dysfunction independently of neurodegenerationBrian R Hoover, Miranda N Reed, Jianjun Su, et al.
Disease Models & Mechanisms|February 8, 2017
The familial dysautonomia disease gene <i>IKBKAP</i> is required in the developing and adult mouse central nervous systemMarta Chaverra, Lynn George, Marc Mergy, et al.
Pageof 8