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International Journal of Biological Sciences|August 4, 2021
Crucial Role of Mammalian Glutaredoxin 3 in Cardiac Energy Metabolism in Diet-induced Obese Mice Revealed by Transcriptome AnalysisNinghui Cheng, Qianxing Mo, Jimmonique Donelson, et al.
Free Radical Biology & Medicine|May 16, 2015
Nuclear glutaredoxin 3 is critical for protection against oxidative stress-induced cell deathKhanh Pham, Rituraj Pal, Ying Qu, et al.
Cell Death & Disease|October 21, 2016
Functional KCa1.1 channels are crucial for regulating the proliferation, migration and differentiation of human primary skeletal myoblastsRajeev B Tajhya, Xueyou Hu, Mark R Tanner, et al.
Science Signaling|October 7, 2025
Phosphorylation of RYR1 at Ser2902 decreases Ca2+ leak in skeletal muscle and susceptibility to malignant hyperthermia and heat strokeRachel Sue Zhen Yee, Chang Seok Lee, Ting Chang, et al.
Circulation. Arrhythmia and Electrophysiology|April 15, 2018
Oxidized CaMKII (Ca2+/Calmodulin-Dependent Protein Kinase II) Is Essential for Ventricular Arrhythmia in a Mouse Model of Duchenne Muscular DystrophyQiongling Wang, Ann P Quick, Shuyi Cao, et al.
American Journal of Physiology. Heart and Circulatory Physiology|October 7, 2022
A microtubule-connexin-43 regulatory link suppresses arrhythmias and cardiac fibrosis in Duchenne muscular dystrophy miceEric Himelman, Julie Nouet, Mauricio A Lillo, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 17, 2022
UBR2 targets myosin heavy chain IIb and IIx for degradation: Molecular mechanism essential for cancer-induced muscle wastingSong Gao, Guohua Zhang, Zicheng Zhang, et al.
Scientific Reports|September 2, 2017
Long-term PGC1β overexpression leads to apoptosis, autophagy and muscle wastingDanesh H Sopariwala, Vikas Yadav, Pierre-Marie Badin, et al.
Developmental Cell|February 19, 2019
YAP Partially Reprograms Chromatin Accessibility to Directly Induce Adult Cardiogenesis In VivoTanner O Monroe, Matthew C Hill, Yuka Morikawa, et al.
Nature Communications|December 4, 2018
Loss of peroxiredoxin-2 exacerbates eccentric contraction-induced force loss in dystrophin-deficient muscleJohn T Olthoff, Angus Lindsay, Reem Abo-Zahrah, et al.
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