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Matrix Biology : Journal of the International Society for Matrix Biology|February 20, 2018
Molecular and tissue alterations of collagens in fibrosisSylvie Ricard-Blum, Georges Baffet, Nathalie ThéretInternational Journal of Hepatology|November 8, 2012
The MAPK MEK1/2-ERK1/2 Pathway and Its Implication in Hepatocyte Cell Cycle ControlJean-Philippe Guégan, Christophe Frémin, Georges BaffetDNA Repair|August 16, 2011
The knock-down of ERCC1 but not of XPF causes multinucleationJulie Rageul, Christophe Frëmin, Frédéric Ezan, et al.Journal of Cellular Physiology|April 12, 2007
MEK/ERK-dependent uPAR expression is required for motility via phosphorylation of P70S6K in human hepatocarcinoma cellsAnne Bessard, Christophe Frémin, Frédéric Ezan, et al.Journal of Cellular Physiology|February 7, 2014
MEK1/2 overactivation can promote growth arrest by mediating ERK1/2-dependent phosphorylation of p70S6KJean-Philippe Guégan, Frédéric Ezan, Luc Gailhouste, et al.Optics Express|June 16, 2015
Linear least square (LLS) method for pixel-resolution analysis of polarization dependent SHG images of collagen fibrilsDenis Rouède, Jean-Jacques Bellanger, Jérémy Bomo, et al.Carcinogenesis|October 9, 2012
MAPK signaling in cisplatin-induced death: predominant role of ERK1 over ERK2 in human hepatocellular carcinoma cellsJean-Philippe Guégan, Frédéric Ezan, Nathalie Théret, et al.American Journal of Physiology. Gastrointestinal and Liver Physiology|March 21, 2003
Regulation and role of p21 and p27 cyclin-dependent kinase inhibitors during hepatocyte differentiation and growthGennady P Ilyin, Denise Glaise, David Gilot, et al.Cancer Research|March 3, 2007
GATA-1 is essential in EGF-mediated induction of nucleotide excision repair activity and ERCC1 expression through ERK2 in human hepatoma cellsLise O Andrieux, Alain Fautrel, Anne Bessard, et al.Hepatology (Baltimore, Md.)|October 24, 2002
PI3K-FRAP/mTOR pathway is critical for hepatocyte proliferation whereas MEK/ERK supports both proliferation and survivalAlexandre Coutant, Claude Rescan, David Gilot, et al.Pageof 4