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Cancer Discovery|November 2, 2014
Mutant KRAS-induced expression of ICAM-1 in pancreatic acinar cells causes attraction of macrophages to expedite the formation of precancerous lesionsGeou-Yarh Liou, Heike Döppler, Brian Necela, et al.
Iscience|May 30, 2023
Inflammatory and alternatively activated macrophages independently induce metaplasia but cooperatively drive pancreatic precancerous lesion growthGeou-Yarh Liou, Alicia K Fleming Martinez, Heike R Döppler, et al.
The Biochemical Journal|February 18, 2010
Induced, selective proteolysis of MLK3 negatively regulates MLK3/JNK signallingGeou-Yarh Liou, Hua Zhang, Eva M Miller, et al.
The Journal of Cell Biology|August 7, 2013
Macrophage-secreted cytokines drive pancreatic acinar-to-ductal metaplasia through NF-κB and MMPsGeou-Yarh Liou, Heike Döppler, Brian Necela, et al.
The Journal of Pathology|June 23, 2017
Glycogen synthase kinase-3β ablation limits pancreatitis-induced acinar-to-ductal metaplasiaLi Ding, Geou-Yarh Liou, Daniel M Schmitt, et al.
Iscience|May 23, 2022
Ym1+ macrophages orchestrate fibrosis, lesion growth, and progression during development of murine pancreatic cancerAlicia K Fleming Martinez, Heike R Döppler, Ligia I Bastea, et al.
The FEBS Journal|September 1, 2020
Macrophages expedite cell proliferation of prostate intraepithelial neoplasia through their downstream target ERKMikalah U Thomas, Justin K Messex, Tu Dang, et al.
Cancers|May 14, 2022
Oncogenic Kras-Mediated Cytokine CCL15 Regulates Pancreatic Cancer Cell Migration and Invasion through ROSJustin K Messex, Kiyah L A Adams, William G Hawkins, et al.
Pancreas|September 14, 2017
Paracrine Secretion of Transforming Growth Factor β by Ductal Cells Promotes Acinar-to-Ductal Metaplasia in Cultured Human Exocrine Pancreas TissuesNaoki Akanuma, Jun Liu, Geou-Yarh Liou, et al.
Cell Reports|March 8, 2016
Mutant KRas-Induced Mitochondrial Oxidative Stress in Acinar Cells Upregulates EGFR Signaling to Drive Formation of Pancreatic Precancerous LesionsGeou-Yarh Liou, Heike Döppler, Kathleen E DelGiorno, et al.
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